• chez wat Oct You're getting warmer

    From Lawyer Daggett@21:1/5 to All on Tue Oct 10 10:41:47 2023
    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where
    new species came from.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to All on Wed Oct 11 17:47:45 2023
    On Tuesday, October 10, 2023 at 1:46:04 PM UTC-4, Lawyer Daggett still can't get the hang of what makes for a good chez watt:

    In the category of a case of begging the question being mistaken for a Chez Watt:

    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where
    new species came from.

    "IDers" is false. Even if the more nearly true word " creationists" is substituted, there are plenty of them who allow for new species or even new genera
    to evolve, by calling it "microevolution." The family Equidae, with tight sequences of
    genera, forces the more intelligent ones into this group.

    The IDer Behe, who has argued *for evolution* (more than I've seen Daggett do)
    goes a step further than most in _Darwin Devolves_. He considers a
    great deal of evolution as being due to breaking genes.

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    If Daggett had done some "spying" to see where the strengths and weaknesses
    of IDers lay, he would have seen an article in Evolution News
    about these broken genes.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of South Carolina
    http://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Thu Oct 12 00:41:40 2023
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Tuesday, October 10, 2023 at 1:46:04?PM UTC-4, Lawyer Daggett still can't >get the hang of what makes for a good chez watt:

    In the category of a case of begging the question being mistaken for a Chez Watt:

    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where
    new species came from.

    "IDers" is false. Even if the more nearly true word " creationists" is >substituted, there are plenty of them who allow for new species or even new genera
    to evolve, by calling it "microevolution." The family Equidae, with tight sequences of
    genera, forces the more intelligent ones into this group.

    The IDer Behe, who has argued *for evolution* (more than I've seen Daggett do)
    goes a step further than most in _Darwin Devolves_. He considers a
    great deal of evolution as being due to breaking genes.


    I know you know Behe calls the consequences of broken genes
    "devolution" in explicit contrast to evolution. In fact and contrary
    to your claim above, Behe claims Darwinian evolution is incapable of
    creating new species.

    Finally, whatever else Daggett might do, he does not make a career of
    posting false and misleading claims against evolution.


    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    If Daggett had done some "spying" to see where the strengths and weaknesses >of IDers lay, he would have seen an article in Evolution News
    about these broken genes.


    EvolutionNews is a biased and unreliable source of the strengths and
    weaknesses of IDers.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Lawyer Daggett@21:1/5 to [email protected] on Thu Oct 12 03:56:38 2023
    On Wednesday, October 11, 2023 at 8:51:05 PM UTC-4, [email protected] wrote:
    On Tuesday, October 10, 2023 at 1:46:04 PM UTC-4, Lawyer Daggett still can't
    get the hang of what makes for a good chez watt:

    In the category of a case of begging the question being mistaken for a Chez Watt:
    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where
    new species came from.


    "IDers" is false. Even if the more nearly true word " creationists" is substituted, there are plenty of them who allow for new species or even new genera
    to evolve, by calling it "microevolution." The family Equidae, with tight sequences of
    genera, forces the more intelligent ones into this group.

    The IDer Behe, who has argued *for evolution* (more than I've seen Daggett do)
    goes a step further than most in _Darwin Devolves_. He considers a
    great deal of evolution as being due to breaking genes.

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    If Daggett had done some "spying" to see where the strengths and weaknesses of IDers lay, he would have seen an article in Evolution News
    about these broken genes.

    Those were words, but they don't seem to include references to IDers who
    "are searching for answers as to how or where new species came from."
    They seem to be more of an, "I don't get the joke." I suggest you don't
    vote for this one. Beyond that, you might take it up with the author.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Thu Oct 12 10:10:37 2023
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Tuesday, October 10, 2023 at 1:46:04?PM UTC-4, Lawyer Daggett still can't >get the hang of what makes for a good chez watt:

    In the category of a case of begging the question being mistaken for a Chez Watt:

    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where
    new species came from.

    "IDers" is false. Even if the more nearly true word " creationists" is >substituted, there are plenty of them who allow for new species or even new genera
    to evolve, by calling it "microevolution." The family Equidae, with tight sequences of
    genera, forces the more intelligent ones into this group.

    The IDer Behe, who has argued *for evolution* (more than I've seen Daggett do)
    goes a step further than most in _Darwin Devolves_. He considers a
    great deal of evolution as being due to breaking genes.

    I know you know Behe calls the consequences of broken genes
    "devolution" in explicit contrast to evolution.

    He is free to choose words that make a point across.
    Where does he explicitly make "evolution" and "devolution"
    contradictory terms.

    The English language is a many-faceted thing where "inflammable"
    and "flammable" are synonymous. Another interesting example
    is the use of "de-escalation" in regard to the Vietnam war,
    even though escalators go in both directions.

    If the New Yorker magazine never published a cartoon showing
    an up-escalator marked "escalator" and a down-escalator marked "de-escalator," its cartoonists weren't on the ball.


    In fact and contrary
    to your claim above, Behe claims Darwinian evolution is incapable of creating new species.

    Documentation, please.


    Finally, whatever else Daggett might do, he does not make a career of posting false and misleading claims against evolution.

    Please identify those IDers who you think are guilty of that, and your grounds for your opinion.


    Concluded in next reply, to be done after I am in my office at the university for office hours.


    Peter Nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Martin Harran@21:1/5 to [email protected] on Thu Oct 12 18:42:59 2023
    On Thu, 12 Oct 2023 10:10:37 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    [�]

    Concluded in next reply, to be done after I am in my office at the university >for office hours.

    So you spend time on Usenet when you are being paid for university
    duties. This is from the guy who claims he is the "goddamn moralizer".

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Lawyer Daggett@21:1/5 to Martin Harran on Thu Oct 12 11:56:05 2023
    On Thursday, October 12, 2023 at 1:46:06 PM UTC-4, Martin Harran wrote:
    On Thu, 12 Oct 2023 10:10:37 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    [匽
    Concluded in next reply, to be done after I am in my office at the university
    for office hours.
    So you spend time on Usenet when you are being paid for university
    duties. This is from the guy who claims he is the "goddamn moralizer".

    That's an ill-informed cheap shot. Sitting around in office hours can be
    a lonely thing. Don't imagine students kept waiting outside the door with
    a "hold on, somebody's wrong on the internet". It's more of waiting around
    in case anybody stops by. If he's composing a post and somebody does,
    he'll gladly drop what he's doing, smile, and see how he can help.

    You ought to apologize.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Thu Oct 12 17:33:16 2023
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    [two lines repeated from first reply, for context]

    Finally, whatever else Daggett might do, he does not make a career of posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's siblings are usually one's biggest competitors for survival. It's hard to guess where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight Felsenstein put in regular appearances in the 1990's. That group went belly-up a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that
    IDers don't try to research the causes of speciation. Perhaps literally, yes, but if one expands "research" to what passes for that [1] in the humanities, then there is ample evidence ripe for the picking from scientific sources.

    [1] You might get quite an argument from Burkhard if you tried to claim
    that their methods are in any way inferior to the methods of research scientists.


    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes
    that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth.

    The following research announcement in Science News from last year talks
    about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm
    "Nearly a hundred genes have been lost during the woolly mammoth's evolution" Date: August 10, 2022
    Source: Stockholm University
    Summary:
    A new study shows that 87 genes have been affected by deletions or short insertions during the course of the mammoth's evolution. The researchers note that their findings have implications for international efforts to resurrect extinct species, including
    the woolly mammoth.


    If Daggett had done some "spying" to see where the strengths and weaknesses >of IDers lay, he would have seen an article in Evolution News
    about these broken genes.


    I'm surprised to see how little you understand the value of reconnaissance, jillery:

    EvolutionNews is a biased and unreliable source of the strengths and weaknesses of IDers.

    So is Wikipedia, even in some strictly scientific contexts. The quality varies greatly in both. This article was reported five days later by Michael Behe and conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of South Carolina
    https://people.math.sc.edu/nyikos

    PS The word "after" that I used in "after I am in my office at the university" took on
    a meaning I neither foresaw nor wanted: I had problems with my laptop that
    our down-the-hall IT person was needed for. You can see that "after" is literally true,
    yet unintentionally misleading.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Martin Harran@21:1/5 to [email protected] on Fri Oct 13 13:49:16 2023
    On Thu, 12 Oct 2023 11:56:05 -0700 (PDT), Lawyer Daggett <[email protected]> wrote:

    On Thursday, October 12, 2023 at 1:46:06?PM UTC-4, Martin Harran wrote:
    On Thu, 12 Oct 2023 10:10:37 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:

    [?
    Concluded in next reply, to be done after I am in my office at the university
    for office hours.
    So you spend time on Usenet when you are being paid for university
    duties. This is from the guy who claims he is the "goddamn moralizer".

    That's an ill-informed cheap shot.

    I don't think it was particularly cheap when it was directed at a
    person who regularly tries to claim the moral high ground on this
    newsgroup yet constantly behaves atrociously towards other posters.

    Sitting around in office hours can be
    a lonely thing. Don't imagine students kept waiting outside the door with
    a "hold on, somebody's wrong on the internet". It's more of waiting around
    in case anybody stops by. If he's composing a post and somebody does,
    he'll gladly drop what he's doing, smile, and see how he can help.

    Neither do I think it was ill informed. I was a lecturer until I
    retired back in 2021 and, whilst I accept that people's mileage
    varies, I never found myself at a loose end in the way you describe
    above. When I had time to spare, I always considered it incumbent on
    me to use that time in some way related to my professional position
    such as additional reading or revising current lectures. Of course
    there were times when I carried out personal business but I confined
    those to things that were either urgent or could not be done outside
    working hours e.g. arranging medical appointments. I certainly would
    never have used my time to engage in hobbies or personal interests

    I note that Peter has said elsewhere that what he posted was
    inadvertently misleading and that he only intended to get his laptop
    sorted out during office hours. I also note, however, that he
    continues to use his title and the name of his university in his
    signature. The institution where I lectured had a very strict policy
    that people should only associate themselves with the institution when
    they were engaged on institution business and never in a situation
    where one was expressing personal views unrelated to the institution.
    I would imagine that the University of South Carolina has a similar
    policy. Peter's inclusion of the weasel words "standard disclaimer"
    does not abrogate his responsibility to conform with that. Indeed,
    they indicate that he is aware that he should not be doing what he is
    doing which really seems just like rather pathetic attempt at creating
    an 'appeal to authority'



    You ought to apologize.


    I don't feel any need whatsoever to apologise to Peter. Where I was
    arguably out of order was that I have said elsewhere that I only
    respond to Peter when he makes unprovoked attacks on me or posts
    bullshit and lies about me. The post I responded to hear was clearly
    not of that nature and it was in a discussion where I wasn't even
    involved. In that context, I regret making the intervention and I
    readily apologise to you for jumping into and potentially derailing a discussion that was essentially between you and him.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Burkhard@21:1/5 to [email protected] on Sat Oct 14 11:16:10 2023
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of posting false and misleading claims against evolution.
    Except, of course, out of ignorance. He thinks it is axiomatic that one's siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that
    IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities, then there is ample evidence ripe for the picking from scientific sources.

    Really, where? And this is a really strange argument for you to make. I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .


    [1] You might get quite an argument from Burkhard if you tried to claim
    that their methods are in any way inferior to the methods of research scientists.


    The following is an instance of Behe's "evolution by devolution"":
    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.
    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2] Behe is quite comfortable with the idea that the great number of genes
    that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth.

    The following research announcement in Science News from last year talks about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm
    "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    Source: Stockholm University
    Summary:
    A new study shows that 87 genes have been affected by deletions or short insertions during the course of the mammoth's evolution. The researchers note that their findings have implications for international efforts to resurrect extinct species,
    including the woolly mammoth.
    If Daggett had done some "spying" to see where the strengths and weaknesses
    of IDers lay, he would have seen an article in Evolution News
    about these broken genes.
    I'm surprised to see how little you understand the value of reconnaissance, jillery:
    EvolutionNews is a biased and unreliable source of the strengths and weaknesses of IDers.
    So is Wikipedia, even in some strictly scientific contexts. The quality varies
    greatly in both. This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.
    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of South Carolina
    https://people.math.sc.edu/nyikos

    PS The word "after" that I used in "after I am in my office at the university" took on
    a meaning I neither foresaw nor wanted: I had problems with my laptop that our down-the-hall IT person was needed for. You can see that "after" is literally true,
    yet unintentionally misleading.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Mon Oct 16 12:05:38 2023
    I am hard pressed to identify anything Harran says about others that
    doesn't apply to himself equally or better.


    On Fri, 13 Oct 2023 13:49:16 +0100, Martin Harran
    <[email protected]> wrote:

    On Thu, 12 Oct 2023 11:56:05 -0700 (PDT), Lawyer Daggett ><[email protected]> wrote:

    On Thursday, October 12, 2023 at 1:46:06?PM UTC-4, Martin Harran wrote:
    On Thu, 12 Oct 2023 10:10:37 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:

    [?
    Concluded in next reply, to be done after I am in my office at the university
    for office hours.
    So you spend time on Usenet when you are being paid for university
    duties. This is from the guy who claims he is the "goddamn moralizer".

    That's an ill-informed cheap shot.

    I don't think it was particularly cheap when it was directed at a
    person who regularly tries to claim the moral high ground on this
    newsgroup yet constantly behaves atrociously towards other posters.

    Sitting around in office hours can be
    a lonely thing. Don't imagine students kept waiting outside the door with
    a "hold on, somebody's wrong on the internet". It's more of waiting around >>in case anybody stops by. If he's composing a post and somebody does,
    he'll gladly drop what he's doing, smile, and see how he can help.

    Neither do I think it was ill informed. I was a lecturer until I
    retired back in 2021 and, whilst I accept that people's mileage
    varies, I never found myself at a loose end in the way you describe
    above. When I had time to spare, I always considered it incumbent on
    me to use that time in some way related to my professional position
    such as additional reading or revising current lectures. Of course
    there were times when I carried out personal business but I confined
    those to things that were either urgent or could not be done outside
    working hours e.g. arranging medical appointments. I certainly would
    never have used my time to engage in hobbies or personal interests

    I note that Peter has said elsewhere that what he posted was
    inadvertently misleading and that he only intended to get his laptop
    sorted out during office hours. I also note, however, that he
    continues to use his title and the name of his university in his
    signature. The institution where I lectured had a very strict policy
    that people should only associate themselves with the institution when
    they were engaged on institution business and never in a situation
    where one was expressing personal views unrelated to the institution.
    I would imagine that the University of South Carolina has a similar
    policy. Peter's inclusion of the weasel words "standard disclaimer"
    does not abrogate his responsibility to conform with that. Indeed,
    they indicate that he is aware that he should not be doing what he is
    doing which really seems just like rather pathetic attempt at creating
    an 'appeal to authority'



    You ought to apologize.


    I don't feel any need whatsoever to apologise to Peter. Where I was
    arguably out of order was that I have said elsewhere that I only
    respond to Peter when he makes unprovoked attacks on me or posts
    bullshit and lies about me. The post I responded to hear was clearly
    not of that nature and it was in a discussion where I wasn't even
    involved. In that context, I regret making the intervention and I
    readily apologise to you for jumping into and potentially derailing a >discussion that was essentially between you and him.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to Burkhard on Tue Oct 17 15:17:27 2023
    On Saturday, October 14, 2023 at 2:21:08 PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.


    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.

    One of the great things about talk.origins is that it helps me to learn all kinds of
    things from unexpected sources. You are an excellent example: by playing in your ballpark, I arrive at
    fresh insights that result from being stimulated in the way that an oyster is stimulated to produce pearls.
    Irritating at first, but often rewarding in the long run.



    [1] You might get quite an argument from Burkhard if you tried to claim that their methods are in any way inferior to the methods of research scientists.

    Looks like I hit the nail on the head with this last remark.


    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2] Behe is quite comfortable with the idea that the great number of genes that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth.

    This is what I was alluding to up there, this time around.

    Here is where Behe wrote about that: https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    This was a result of Behe researching some of the latest scientific news:

    The following research announcement in Science News from last year talks about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm
    "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    Source: Stockholm University
    Summary:
    A new study shows that 87 genes have been affected by deletions or short insertions during the course of the mammoth's evolution. The researchers note that their findings have implications for international efforts to resurrect extinct species,
    including the woolly mammoth.

    <snip for focus>

    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of So. Carolina in Columbia
    https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Burkhard@21:1/5 to [email protected] on Wed Oct 18 00:18:09 2023
    On Wednesday, October 18, 2023 at 12:21:11 AM UTC+2, [email protected] wrote:
    On Saturday, October 14, 2023 at 2:21:08 PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?
    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.


    Which humanities method is used in that research? I can't see any.
    And what documents do you mean? If you simply mean that rather
    than doing his own experiments, he uses those made by other, sorry,
    no, mere "displaying reading ability" does not make this into humanities-type research.

    Humanities-type ID research on gene-breakages should form (testable)
    hypothesis of the type: The designer wanted these genera because....
    or The designer chose this method of gene breakages because...

    And this is a really strange argument for you to make.
    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]
    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .
    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.

    One of the great things about talk.origins is that it helps me to learn all kinds of
    things from unexpected sources. You are an excellent example: by playing in your ballpark, I arrive at
    fresh insights that result from being stimulated in the way that an oyster is stimulated to produce pearls.
    Irritating at first, but often rewarding in the long run.

    [1] You might get quite an argument from Burkhard if you tried to claim that their methods are in any way inferior to the methods of research scientists.
    Looks like I hit the nail on the head with this last remark.

    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken >that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth.
    This is what I was alluding to up there, this time around.

    Here is where Behe wrote about that: https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    This was a result of Behe researching some of the latest scientific news:
    The following research announcement in Science News from last year talks about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm
    "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    Source: Stockholm University
    Summary:
    A new study shows that 87 genes have been affected by deletions or short insertions during the course of the mammoth's evolution. The researchers note that their findings have implications for international efforts to resurrect extinct species,
    including the woolly mammoth.
    <snip for focus>
    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.
    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?
    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of So. Carolina in Columbia
    https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Wed Oct 18 12:00:50 2023
    On Tue, 17 Oct 2023 15:17:27 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Saturday, October 14, 2023 at 2:21:08?PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06?AM UTC+2, [email protected] wrote: >> > On Thursday, October 12, 2023 at 12:46:05?AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of >> > > posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's >> > siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that
    IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.


    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.

    One of the great things about talk.origins is that it helps me to learn all kinds of
    things from unexpected sources. You are an excellent example: by playing in your ballpark, I arrive at
    fresh insights that result from being stimulated in the way that an oyster is stimulated to produce pearls.
    Irritating at first, but often rewarding in the long run.



    [1] You might get quite an argument from Burkhard if you tried to claim >> > that their methods are in any way inferior to the methods of research scientists.

    Looks like I hit the nail on the head with this last remark.


    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes
    that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth.

    This is what I was alluding to up there, this time around.

    Here is where Behe wrote about that: >https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    This was a result of Behe researching some of the latest scientific news:

    The following research announcement in Science News from last year talks >> > about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm
    "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    Source: Stockholm University
    Summary:
    A new study shows that 87 genes have been affected by deletions or short insertions during the course of the mammoth's evolution. The researchers note that their findings have implications for international efforts to resurrect extinct species,
    including the woolly mammoth.

    <snip for focus>

    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?



    I did you and Behe a favor, to not respond to your post. After all, I
    wouldn't want some troll to mindlessly accuse me of "bickering".

    I'm not surprised you fell for this latest Behe nonsense. Of course
    mutations are "very much more likely to degrade genetic features than
    to construct new ones". Anybody who stayed awake in biology knows
    this already. What Behe and you and other cdesign proponentsists
    always conveniently forget to mention is that genetic features which
    improve organisms' fitness are "very much more likely" to be amplified
    via reproduction and natural selection.

    Stipulating for argument's sake everything Behe says is technically
    correct, he still conveniently forgets to mention all the
    "constructive" mutations his example organisms almost certainly
    evolved during the same time period. It's called cherrypicking, a
    typical tactic of Creationists.

    However, Behe is factually incorrect to presume devolved "genes are
    gone forever". Even when genes have zero function, they will likely
    remain in the population for millions of years, as part of the junk
    gene collection cdesign proponentsists are so fond of denying. During
    that time, those functionless genes will continue to mutate, possibly
    to produce an entirely new function.

    And since I mention new functions, Behe is fond of saying devolved
    genes "don’t explain how the functioning genes got there in the first
    place. Well guess what? ID doesn't explain it either. In fact,
    cdesign proponentsists make zero effort to explain which genes their presumptive designer makes, nevermind how.

    In summary, your cited article, and your post, are just more mindless
    cdesign proponentsist noise.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to Burkhard on Fri Oct 20 17:25:27 2023
    On Wednesday, October 18, 2023 at 3:21:12 AM UTC-4, Burkhard wrote:
    On Wednesday, October 18, 2023 at 12:21:11 AM UTC+2, [email protected] wrote:
    On Saturday, October 14, 2023 at 2:21:08 PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of
    posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.

    Which humanities method is used in that research?

    Trying to find all relevant documents, and to study them in depth,
    and to weave what one has studied into a coherent hypothesis.

    Behe did this to a much greater extent in _The Edge of Evolution_. He used malaria
    as an example of "trench warfare" between two antagonists [the malaria parasite and the human host]
    where lasting victory seems to be a matter of having at least three different mutations
    in the parasite, [with us humans it is different -- see below] all of which are beneficial
    under the circumstances dictated by the environment.

    With the sickle cell trait -- NOT the disease [1] -- the parasite has been stymied; in contrast,
    one drug treatment after another has been successfully countered by it.

    I wonder whether Bill Rogers, our malaria professional, can either (1) find flaws with it or
    (2) has ever composed a more masterly account than the mere amateur, Michael Behe, has done.

    [1] The sickle cell trait is characterized as having one normal gene
    and one mutated version of it that protects against malaria.
    The disease only has the mutated version. It protects against malaria even a tad better,
    but it results in a debilitating anemia that is even worse than most cases of malaria.


    I can't see any.

    I haven't provided any until now. This is an ongoing discussion in which I don't
    try to anticipate your every criticism.

    And what documents do you mean? If you simply mean that rather
    than doing his own experiments,

    Just what "experiments" is someone trying to discover the author of
    the Voinich manuscript supposed to do? or someone trying to figure
    out the hour in which the climactic Norman assault at Hastings took place?


    he uses those made by other, sorry,
    no, mere "displaying reading ability" does not make this into humanities-type
    research.

    See what I wrote about Behe and malaria. He's gone into lots of angles that one does
    not find in standard treatments of the subject, or in the writings of any single author.
    Certainly not in the writings of Bill Rogers.

    Humanities-type ID research on gene-breakages should form (testable) hypothesis of the type: The designer wanted these genera because....

    A creationist would frame such hypotheses, but Behe allows for lots of
    unguided evolution. So his hypotheses are of a different sort, like the
    one I mentioned above: how likely is that unguided processes will
    arrive at a successful counter-measure of the parasite to the entrapment
    and suffocation the sickle cell trait "uses" to kill the parasites?

    A vast period of time involving several million years might overcome the odds, but neither humans nor the parasite have been around that long.
    Humans, on the other hand, needed only ONE mutation in ONE human with
    several offspring to produce the sickle cell trait. Consequently, Behe doesn't claim
    that the mutation was divinely caused or even that the Designer foresaw it.

    or The designer chose this method of gene breakages because...

    No, gene breakages are a purely Darwinian means of producing whole new species. Behe made that point right in the article I linked for jillery:

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/


    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    <crickets>

    You didn't say anything more this time around, but I've left in some of what you left in,
    with one comment about what you did say this time around.

    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.

    <snip for focus>

    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken >that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth.
    This is what I was alluding to up there, this time around.
    <small snip>

    This was a result of Behe researching some of the latest scientific news:

    The following research announcement in Science News from last year talks
    about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm
    "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    <snip for focus>

    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here: it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?

    You made a weak stab at it this time, but you may add more in the light of what I wrote this time.
    Such back-and-forth is what Usenet was designed for.


    Peter Nyikos
    Professor, Dept. of Mathematics
    U. of So. Carolina -- standard disclaimer-- https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Fri Oct 20 18:32:05 2023
    I've spent a lot of time in sci.bio.paleontology today, but the reply I did to your fan Burkhard
    just now, and this reply to you, jillery, is all the time I can spare for talk.origins today.
    And this means no more posts until Monday, if my weekend break proceeds uninterrupted,
    as it has all through 2023 so far.

    On Wednesday, October 18, 2023 at 12:06:12 PM UTC-4, jillery wrote:
    On Tue, 17 Oct 2023 15:17:27 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Saturday, October 14, 2023 at 2:21:08?PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06?AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05?AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of >> > > posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that >> > IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.


    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.

    I snipped the following from my reply to Burkhard, for the sake of staying in focus:

    One of the great things about talk.origins is that it helps me to learn all kinds of
    things from unexpected sources. You are an excellent example: by playing in your ballpark, I arrive at
    fresh insights that result from being stimulated in the way that an oyster is stimulated to produce pearls.
    Irritating at first, but often rewarding in the long run.



    [1] You might get quite an argument from Burkhard if you tried to claim >> > that their methods are in any way inferior to the methods of research scientists.

    Looks like I hit the nail on the head with this last remark.

    <snip to get to the following link:>

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time >> > to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?

    I did you and Behe a favor, to not respond to your post. After all, I wouldn't want some troll to mindlessly accuse me of "bickering".

    I can't think of anyone besides Martin Harran who would stoop so low.
    I have more weighty things to talk about.


    I'm not surprised you fell for this latest Behe nonsense.

    This is very mindful false advertising.


    Of course
    mutations are "very much more likely to degrade genetic features than
    to construct new ones". Anybody who stayed awake in biology knows
    this already.

    But NOT what Behe said about it: they are able to produce new species
    in a strictly Darwinian fashion, as opposed to producing IC structures by a direct method.

    IOW, Behe has illustrated exactly what he said on page 40 of "indirect, circuitous methods."
    What's left of hair production genes in the woolly mammoth may well have an IC system that
    produces great volumes of hair that the previously unbroken genes had suppressed the
    production of, in the interglacial and pre-glaciation warm periods.



    What Behe and you and other [snip obsolete, irrelevant term on which you are fixated]
    always conveniently forget to mention is that genetic features which
    improve organisms' fitness are "very much more likely" to be amplified
    via reproduction and natural selection.

    That's because that is belaboring the obvious. The issue here is that some features that improve organisms' fitness in warm periods of earth
    history become deleterious in colder periods. Besides hair, woolly mammoths produced lots of insulating fat as genes which suppressed its formation became broken.



    Stipulating for argument's sake everything Behe says is technically
    correct, he still conveniently forgets to mention all the
    "constructive" mutations his example organisms almost certainly
    evolved during the same time period.

    And you conveniently "forget" to supply us with any specific examples.


    It's called cherrypicking, a
    typical tactic of Creationists.

    And of anti-creationists who know less science than Behe does
    about, say, malaria [see my reply to Burkhard] and cherry-pick
    with abandon from my posts, from time to time. One of the worst,
    if not THE worst, has killfiled you.

    However, Behe is factually incorrect to presume devolved "genes are
    gone forever".

    Minor exaggerations like this are nothing compared to the
    exaggeration you are fixated on. Especially when you
    falsely accuse me of it, as you did above.


    Even when genes have zero function, they will likely
    remain in the population for millions of years, as part of the junk
    gene collection <snip your fixation> are so fond of denying. During
    that time, those functionless genes will continue to mutate, possibly
    to produce an entirely new function.

    Once in a million blue moons. [If that's an exaggeration, prove it!]


    And since I mention new functions, Behe is fond of saying devolved
    genes "don’t explain how the functioning genes got there in the first place. Well guess what? ID doesn't explain it either.

    It does in the case of DP, which may become strongly supported
    as we send probes to earth-like exoplanets.


    In fact, <snip your fixation>
    make zero effort to explain which genes their
    presumptive designer makes, nevermind how.

    There is some effort made in _The Edge of Evolution_ to find genes
    that might have been designed. But the bacterial flagellum is still the gold standard in that respect.
    And DP is one hypothesis for how it might have been designed by
    beings no more intelligent than Homo sapiens.


    In summary, your cited article, and your post, are

    ...misrepresented by you, in the part I snipped here, to be like what our initially science-based discussions most often devolve into:
    jillery-generated mindless noise.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    Univ. of So. Carolina at Columbia
    http://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Burkhard@21:1/5 to [email protected] on Sat Oct 21 14:30:15 2023
    On Saturday, October 21, 2023 at 2:26:14 AM UTC+2, [email protected] wrote:
    On Wednesday, October 18, 2023 at 3:21:12 AM UTC-4, Burkhard wrote:
    On Wednesday, October 18, 2023 at 12:21:11 AM UTC+2, [email protected] wrote:
    On Saturday, October 14, 2023 at 2:21:08 PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of
    posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that
    IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage? If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.

    Which humanities method is used in that research?
    Trying to find all relevant documents, and to study them in depth,
    and to weave what one has studied into a coherent hypothesis.

    That is not distinctive for humanities, that is just research synthesis,
    and is something that all disciplines do do - hence "standing on the shoulders of giants". Of course researchers don't have to do all
    experiments on their own, they also read the results of others, and try to build
    theories that account for all of them.

    And in the natural sciences, meta-analysis is a rigorous research method, with the Cochrane review setting the gold standard.


    Behe did this to a much greater extent in _The Edge of Evolution_. He used malaria
    as an example of "trench warfare" between two antagonists [the malaria parasite and the human host]
    where lasting victory seems to be a matter of having at least three different mutations
    in the parasite, [with us humans it is different -- see below] all of which are beneficial
    under the circumstances dictated by the environment.

    I can't see any research methodology that is distinctive for the humanities there,
    this is just totally normal theory formation in the natural sciences.


    With the sickle cell trait -- NOT the disease [1] -- the parasite has been stymied; in contrast,
    one drug treatment after another has been successfully countered by it.

    I wonder whether Bill Rogers, our malaria professional, can either (1) find flaws with it or
    (2) has ever composed a more masterly account than the mere amateur, Michael Behe, has done.

    [1] The sickle cell trait is characterized as having one normal gene
    and one mutated version of it that protects against malaria.
    The disease only has the mutated version. It protects against malaria even a tad better,
    but it results in a debilitating anemia that is even worse than most cases of malaria.
    I can't see any.
    I haven't provided any until now. This is an ongoing discussion in which I don't
    try to anticipate your every criticism.
    And what documents do you mean? If you simply mean that rather
    than doing his own experiments,
    Just what "experiments" is someone trying to discover the author of
    the Voinich manuscript supposed to do?

    First, that misunderstands the argument entirely. Or are you really
    arguing that if A is lacking a trait X, and B is lacking the same trait X,
    A is B? That is rather obviously fallacious. Footballers don't use javelins, and chess players don't use javelins, but that does not mean that
    football is the same as chess.

    Even if it were true that humanities researchers do not carry out experiments, the fact that Behe also does not carry out experiments would not make him
    a humanities researcher, not by a long mile.

    But apart from being logically flawed, it's also wrong as a matter of fact. Of course
    Voynich researchers have and are carrying our experiments.Gregory Hodgins e.g. carried out a carbon dating analysis, that rules out several theories that treated
    it as a modern fake. The ink has equally been subjected to testing, as were the
    paint in the colourings (Barabe, Joseph G. (1 April 2009).
    "Materials Analysis of the Voynich Manuscript") - showing e.g. that text and images
    are contemporaneous, and that the author had access to some more
    unusual materials.






    or someone trying to figure
    out the hour in which the climactic Norman assault at Hastings took place?
    he uses those made by other, sorry,
    no, mere "displaying reading ability" does not make this into humanities-type
    research.
    See what I wrote about Behe and malaria. He's gone into lots of angles that one does
    not find in standard treatments of the subject, or in the writings of any single author.
    Certainly not in the writings of Bill Rogers.

    And that turns it into humanities research how?

    Humanities-type ID research on gene-breakages should form (testable) hypothesis of the type: The designer wanted these genera because....
    A creationist would frame such hypotheses, but Behe allows for lots of unguided evolution.

    But he also argues design, and a design theory would have to use
    these hypothesis somewhere

    So his hypotheses are of a different sort, like the
    one I mentioned above: how likely is that unguided processes will
    arrive at a successful counter-measure of the parasite to the entrapment
    and suffocation the sickle cell trait "uses" to kill the parasites?

    That quantifies over all processes, including unknown ones. That is
    just bad research methodology. Doing natural sciences badly does not
    mean doing it like the humanities do



    A vast period of time involving several million years might overcome the odds,
    but neither humans nor the parasite have been around that long.
    Humans, on the other hand, needed only ONE mutation in ONE human with several offspring to produce the sickle cell trait. Consequently, Behe doesn't claim
    that the mutation was divinely caused or even that the Designer foresaw it.

    So why then is it a design theory? Me running into a door which causes, unforeseen by me, to smash a mirror on the other side does not make
    me the designer of the crashed mirror

    or The designer chose this method of gene breakages because...
    No, gene breakages are a purely Darwinian means of producing whole new species. Behe made that point right in the article I linked for jillery:

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/
    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]
    <crickets>

    nothing worth commenting on, really


    You didn't say anything more this time around, but I've left in some of what you left in,
    with one comment about what you did say this time around.
    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.
    <snip for focus>
    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken >that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes
    that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth. This is what I was alluding to up there, this time around.
    <small snip>
    This was a result of Behe researching some of the latest scientific news:

    The following research announcement in Science News from last year talks
    about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    <snip for focus>

    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here: it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?
    You made a weak stab at it this time, but you may add more in the light of what I wrote this time.
    Such back-and-forth is what Usenet was designed for.
    Peter Nyikos
    Professor, Dept. of Mathematics
    U. of So. Carolina -- standard disclaimer-- https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Sun Oct 22 03:27:39 2023
    On Fri, 20 Oct 2023 17:25:27 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    No, gene breakages are a purely Darwinian means of producing whole new >species. Behe made that point right in the article I linked for jillery:

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/


    Incorrect. Behe made no such claim. What Behe claims above is that
    gene breakage is "the most likely way" organisms evolve, and so "can’t account for the origins of sophisticated biological systems." He said
    nothing about creating whole new species, or that gene breakage is
    limited to Darwinian means. In fact, gene breakage is entirely
    consistent with ID as well.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to say Behe on Sun Oct 22 03:43:51 2023
    On Fri, 20 Oct 2023 18:32:05 -0700 (PDT), "[email protected]" <[email protected]> trolled:

    Despite peter2's professed lack of time, it's remarkable how he still
    managed to find time to post even more mindless obfuscating noise.


    I've spent a lot of time in sci.bio.paleontology today, but the reply I did to your fan Burkhard
    just now, and this reply to you, jillery, is all the time I can spare for talk.origins today.
    And this means no more posts until Monday, if my weekend break proceeds uninterrupted,
    as it has all through 2023 so far.

    On Wednesday, October 18, 2023 at 12:06:12?PM UTC-4, jillery wrote:
    On Tue, 17 Oct 2023 15:17:27 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:

    On Saturday, October 14, 2023 at 2:21:08?PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06?AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05?AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of
    posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has
    to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that >> >> > IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.


    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.

    I snipped the following from my reply to Burkhard, for the sake of staying in focus:


    Not sure how "the following" does anything to help anybody stay in
    focus here.


    One of the great things about talk.origins is that it helps me to learn all kinds of
    things from unexpected sources. You are an excellent example: by playing in your ballpark, I arrive at
    fresh insights that result from being stimulated in the way that an oyster is stimulated to produce pearls.
    Irritating at first, but often rewarding in the long run.



    [1] You might get quite an argument from Burkhard if you tried to claim
    that their methods are in any way inferior to the methods of research scientists.

    Looks like I hit the nail on the head with this last remark.

    <snip to get to the following link:>

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here: >> >> > it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time >> >> > to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?

    I did you and Behe a favor, to not respond to your post. After all, I
    wouldn't want some troll to mindlessly accuse me of "bickering".

    I can't think of anyone besides Martin Harran who would stoop so low.
    I have more weighty things to talk about.


    I'm not surprised you fell for this latest Behe nonsense.

    This is very mindful false advertising.


    Your "this" is a fact.


    Of course
    mutations are "very much more likely to degrade genetic features than
    to construct new ones". Anybody who stayed awake in biology knows
    this already.

    But NOT what Behe said about it: they are able to produce new species
    in a strictly Darwinian fashion, as opposed to producing IC structures by a direct method.


    What I quoted came directly from your cited article. OTOH what you
    say Behe said in it is a figment of your imagination.


    IOW, Behe has illustrated exactly what he said on page 40 of "indirect, circuitous methods."
    What's left of hair production genes in the woolly mammoth may well have an IC system that
    produces great volumes of hair that the previously unbroken genes had suppressed the
    production of, in the interglacial and pre-glaciation warm periods.



    What Behe and you and other cdesign proponentsists
    always conveniently forget to mention is that genetic features which
    improve organisms' fitness are "very much more likely" to be amplified
    via reproduction and natural selection.

    That's because that is belaboring the obvious. The issue here is that some >features that improve organisms' fitness in warm periods of earth
    history become deleterious in colder periods. Besides hair, woolly mammoths >produced lots of insulating fat as genes which suppressed its formation became broken.


    Yes, that's how natural selection works, yet another fact that anybody
    who stayed awake in biology already knows.


    Stipulating for argument's sake everything Behe says is technically
    correct, he still conveniently forgets to mention all the
    "constructive" mutations his example organisms almost certainly
    evolved during the same time period.

    And you conveniently "forget" to supply us with any specific examples.


    Are you denying that beneficial mutations evolve? That's more than
    Behe does.


    It's called cherrypicking, a typical tactic of Creationists.

    And of anti-creationists who know less science than Behe does
    about, say, malaria [see my reply to Burkhard] and cherry-pick
    with abandon from my posts, from time to time. One of the worst,
    if not THE worst, has killfiled you.

    However, Behe is factually incorrect to presume devolved "genes are
    gone forever".

    Minor exaggerations like this are nothing compared to the
    exaggeration you are fixated on. Especially when you
    falsely accuse me of it, as you did above.


    I'm not surprised you conveniently forgot to identify what is that "exaggeration" that allegedly fixates me.


    Even when genes have zero function, they will likely
    remain in the population for millions of years, as part of the junk
    gene collection cdesign proponenentsists are so fond of denying. During
    that time, those functionless genes will continue to mutate, possibly
    to produce an entirely new function.

    Once in a million blue moons. [If that's an exaggeration, prove it!]


    Are you denying that possibility? That's more than Behe does.


    And since I mention new functions, Behe is fond of saying devolved
    genes "don’t explain how the functioning genes got there in the first
    place. Well guess what? ID doesn't explain it either.

    It does in the case of DP, which may become strongly supported
    as we send probes to earth-like exoplanets.


    If only Behe had mentioned DP. Since he hadn't, your comment is just
    more obfuscating noise.


    In fact, cdesign proponentsists
    make zero effort to explain which genes their
    presumptive designer makes, nevermind how.

    There is some effort made in _The Edge of Evolution_ to find genes
    that might have been designed.


    Perhaps I missed it. Cite your "some effort".


    But the bacterial flagellum is still the gold standard in that respect.
    And DP is one hypothesis for how it might have been designed by
    beings no more intelligent than Homo sapiens.


    In summary, your cited article, and your post, are

    ...misrepresented by you, in the part I snipped here, to be like what our >initially science-based discussions most often devolve into: >jillery-generated mindless noise.


    Yeah, I get that a lot from willfully stupid trolls.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Martin Harran@21:1/5 to [email protected] on Sun Oct 22 22:13:27 2023
    On Fri, 20 Oct 2023 18:32:05 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    [...]

    I can't think of anyone besides Martin Harran who would stoop so low.

    I now accept that trying to get you to recognise your obnoxious
    behaviour is a waste of time. Either:

    a) You are totally incapable of recognising how obnoxious it is

    or b) You simply cannot stop yourself from behaving obnoxiously.

    Either way, behaving as a decent, civilised person is obviously
    totally beyond your capability so trying to get you to change is a
    totally useless exercise.


    [...]

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Bob Casanova@21:1/5 to All on Sun Oct 22 15:02:36 2023
    On Sun, 22 Oct 2023 22:13:27 +0100, the following appeared
    in talk.origins, posted by Martin Harran
    <[email protected]>:

    On Fri, 20 Oct 2023 18:32:05 -0700 (PDT), "[email protected]" ><[email protected]> wrote:

    [...]

    I can't think of anyone besides Martin Harran who would stoop so low.

    I now accept that trying to get you to recognise your obnoxious
    behaviour is a waste of time. Either:

    a) You are totally incapable of recognising how obnoxious it is

    or b) You simply cannot stop yourself from behaving obnoxiously.

    Either way, behaving as a decent, civilised person is obviously
    totally beyond your capability so trying to get you to change is a
    totally useless exercise.

    I'd simply note that a) and b) are not mutually exclusive;
    in fact, they're often related. Or in simpler terms,
    "Embrace the value of 'and' ".

    As for the remainder, well, I'm happy that you finally
    accepted reality. :-)

    --

    Bob C.

    "The most exciting phrase to hear in science,
    the one that heralds new discoveries, is not
    'Eureka!' but 'That's funny...'"

    - Isaac Asimov

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Sun Oct 22 22:55:10 2023
    On Sun, 22 Oct 2023 22:13:27 +0100, Martin Harran
    <[email protected]> wrote:

    On Fri, 20 Oct 2023 18:32:05 -0700 (PDT), "[email protected]" ><[email protected]> wrote:

    [...]

    I can't think of anyone besides Martin Harran who would stoop so low.

    I now accept that trying to get you to recognise your obnoxious
    behaviour is a waste of time. Either:

    a) You are totally incapable of recognising how obnoxious it is

    or b) You simply cannot stop yourself from behaving obnoxiously.

    Either way, behaving as a decent, civilised person is obviously
    totally beyond your capability so trying to get you to change is a
    totally useless exercise.


    Mirror.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Tue Oct 24 14:30:19 2023
    This has been another super-busy day, so I'm posting only in reply
    to posts with comments that are very much on-topic for the purposes
    for which talk.origins and sci.bio.evolution were set up, and reciprocating.

    And since s.b.e. went extinct over a decade ago, its on-topic
    themes have been picked up by talk.origins and sci.bio.paleontology.

    On Sunday, October 22, 2023 at 3:31:15 AM UTC-4, jillery wrote:
    On Fri, 20 Oct 2023 17:25:27 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    On Wednesday, October 18, 2023 at 3:21:12 AM UTC-4, Burkhard wrote:

    Humanities-type ID research on gene-breakages should form (testable)
    hypothesis of the type:
    [...]
    The designer chose this method of gene breakages because...

    No, gene breakages are a purely Darwinian means of producing whole new >species. Behe made that point right in the article I linked for jillery:

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Incorrect. Behe made no such claim.

    Not that you deserve it, but I'll give you the benefit of the doubt,
    and assume that you did not read as far as the last paragraph.
    Note the word "devolution" which Behe uses to mean "evolution that results from
    degradation of the genome."

    "At least in retrospect, it’s easy to see that devolution must happen — for the simple reason that helpful degradative mutations are more plentiful than helpful constructive ones and thus arrive more quickly for natural selection to multiply. The
    more recent results recounted here just pile more evidence onto that gathered in _Darwin_Devolves_ showing Darwin’s mechanism is powerfully devolutionary. That simple realization neatly explains results ranging from the evolutionary behavior of yeast
    in a comfy modern laboratory, to the speciation of megafauna in raw nature millions of years ago, and almost certainly to everything in between."

    Note the word "speciation" in that last half sentence.

    What Behe claims above is that
    gene breakage is "the most likely way" organisms evolve, and so "can’t account for the origins of sophisticated biological systems."

    Those are on the level of whole new families or orders,
    but Behe stopped with speciation above.


    He said nothing about creating whole new species,

    i.e., speciation -- but he did talk about that "loud and clear,"
    as the quoted comments show.


    or that gene breakage is
    limited to Darwinian means. In fact, gene breakage is entirely
    consistent with ID as well.

    Yes, but Burkhard was talking about divining the motivations of
    (possibly human-level) designers, and in the case of the breakages Behe is talking about,
    the Laplacian formula applies, "Sire, I had no need of that hypothesis."


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    Univ. of So. Carolina in Columbia
    http://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to Burkhard on Tue Oct 24 15:24:25 2023
    On Saturday, October 21, 2023 at 5:31:15 PM UTC-4, Burkhard wrote:
    On Saturday, October 21, 2023 at 2:26:14 AM UTC+2, [email protected] wrote:
    On Wednesday, October 18, 2023 at 3:21:12 AM UTC-4, Burkhard wrote:
    On Wednesday, October 18, 2023 at 12:21:11 AM UTC+2, [email protected] wrote:
    On Saturday, October 14, 2023 at 2:21:08 PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of
    posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that
    IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.

    Which humanities method is used in that research?

    Trying to find all relevant documents, and to study them in depth,
    and to weave what one has studied into a coherent hypothesis.

    That is not distinctive for humanities, that is just research synthesis,
    and is something that all disciplines do do - hence "standing on the shoulders of giants". Of course researchers don't have to do all
    experiments on their own, they also read the results of others, and try to build
    theories that account for all of them.

    That is exactly what Behe does with malaria in _The Edge of Evolution_, as I had written below.
    Gene breakage is a much newer topic for him, so not on the same high level yet,
    but his approach is promising.

    And in the natural sciences, meta-analysis is a rigorous research method, with
    the Cochrane review setting the gold standard.

    Behe did this to a much greater extent in _The Edge of Evolution_. He used malaria
    as an example of "trench warfare" between two antagonists [the malaria parasite and the human host]
    where lasting victory seems to be a matter of having at least three different mutations
    in the parasite, [with us humans it is different -- see below] all of which are beneficial
    under the circumstances dictated by the environment.

    I can't see any research methodology that is distinctive for the humanities there,
    this is just totally normal theory formation in the natural sciences.

    Yes, but I've been making the point that the research Behe does IS real research
    if we use the standards of the humanities for the word "research."


    With the sickle cell trait -- NOT the disease [1] -- the parasite has been stymied; in contrast,
    one drug treatment after another has been successfully countered by it.

    I wonder whether Bill Rogers, our malaria professional, can either (1) find flaws with it or
    (2) has ever composed a more masterly account than the mere amateur, Michael Behe, has done.

    [1] The sickle cell trait is characterized as having one normal gene
    and one mutated version of it that protects against malaria.
    The disease only has the mutated version. It protects against malaria even a tad better,
    but it results in a debilitating anemia that is even worse than most cases of malaria.

    I can't see any.

    I haven't provided any until now. This is an ongoing discussion in which I don't
    try to anticipate your every criticism.


    And what documents do you mean? If you simply mean that rather
    than doing his own experiments,

    Just what "experiments" is someone trying to discover the author of
    the Voinich manuscript supposed to do?

    First, that misunderstands the argument entirely. Or are you really
    arguing that if A is lacking a trait X, and B is lacking the same trait X,
    A is B?

    Obviously not! Behe looks at numerous measures and counter-measures
    in this never-ending "trench warfare." There are numerous types of
    traits evolving on either side.


    That is rather obviously fallacious. Footballers don't use javelins,
    and chess players don't use javelins, but that does not mean that
    football is the same as chess.

    Why do you make these analogies in the absence of any palpable
    statement by me to which they could be applicable?


    Even if it were true that humanities researchers do not carry out experiments,
    the fact that Behe also does not carry out experiments would not make him
    a humanities researcher, not by a long mile.

    Obviously not! I gave the main point I was making above:
    "Yes, but I've been making the point..." etc.

    But apart from being logically flawed, it's also wrong as a matter of fact. Of course
    Voynich researchers have and are carrying our experiments.Gregory Hodgins e.g.
    carried out a carbon dating analysis, that rules out several theories that treated
    it as a modern fake.

    Point taken. But Behe is standing on the shoulders of giants
    for his dating of when "Sickle Trait Eve" was born, and it's
    quite important as to being able to show that the malaria parasite
    has had all this time to mutate a defense, and it has failed to do so.

    The ink has equally been subjected to testing, as were the
    paint in the colourings (Barabe, Joseph G. (1 April 2009).
    "Materials Analysis of the Voynich Manuscript") - showing e.g. that text and images
    are contemporaneous, and that the author had access to some more
    unusual materials.


    or someone trying to figure
    out the hour in which the climactic Norman assault at Hastings took place?

    Let me know when you figure out an experiment that would
    shed light on this at this late date.

    he uses those made by other, sorry,
    no, mere "displaying reading ability" does not make this into humanities-type
    research.

    See what I wrote about Behe and malaria. He's gone into lots of angles that one does
    not find in standard treatments of the subject, or in the writings of any single author.
    Certainly not in the writings of Bill Rogers.

    And that turns it into humanities research how?

    Missing the main point again.


    Humanities-type ID research on gene-breakages should form (testable) hypothesis of the type: The designer wanted these genera because....
    A creationist would frame such hypotheses, but Behe allows for lots of unguided evolution.


    But he also argues design, and a design theory would have to use
    these hypothesis somewhere.

    Yes...in very carefully chosen cases that call for it. A good design
    theorist does not go hunting for design under every bed, as the metaphor goes.

    So his hypotheses are of a different sort, like the
    one I mentioned above: how likely is that unguided processes will
    arrive at a successful counter-measure of the parasite to the entrapment and suffocation the sickle cell trait "uses" to kill the parasites?


    That quantifies over all processes, including unknown ones.

    That is why it is so important that the sickle cell trait has been
    around for thousands of years. In contrast, the usual drug based
    treatments get mutated around every few years, because of
    the staggering number of individual parasites in every infected person.


    That is
    just bad research methodology.

    No worse than quantifying over all the unknown people that
    produced the ancient Indus Valley script. Despite about
    a century and a half of effort, no one has deciphered a single sentence.


    Doing natural sciences badly does not
    mean doing it like the humanities do

    A vast period of time involving several million years might overcome the odds,
    but neither humans nor the parasite have been around that long.
    Humans, on the other hand, needed only ONE mutation in ONE human with several offspring to produce the sickle cell trait. Consequently, Behe doesn't claim
    that the mutation was divinely caused or even that the Designer foresaw it.

    So why then is it a design theory?

    The theory has to do with how many successive neutral or slightly deleterious mutations it takes to evolve a very precise certain feature, before "design"
    is hypothesized. I'm rushed for time, but IIRC Behe puts "the edge of evolution" at "more than three."


    <snip for focus>

    or The designer chose this method of gene breakages because...

    No, gene breakages are a purely Darwinian means of producing whole new species. Behe made that point right in the article I linked for jillery:

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/
    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    <crickets>

    nothing worth commenting on, really

    Then you've lost the "strange argument" game on your home turf. Would you like a rematch?


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of So. Carolina at Columbia
    http://people.math.sc.edu/nyikos

    PS you had nothing new to say below, but I am pressed for time and haven't commented on what's there either. But I left it in below, intact.

    You didn't say anything more this time around, but I've left in some of what you left in,
    with one comment about what you did say this time around.
    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.
    <snip for focus>
    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes
    that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth. This is what I was alluding to up there, this time around.
    <small snip>
    This was a result of Behe researching some of the latest scientific news:

    The following research announcement in Science News from last year talks
    about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    <snip for focus>

    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?
    You made a weak stab at it this time, but you may add more in the light of what I wrote this time.
    Such back-and-forth is what Usenet was designed for.
    Peter Nyikos
    Professor, Dept. of Mathematics
    U. of So. Carolina -- standard disclaimer-- https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From *Hemidactylus*@21:1/5 to [email protected] on Tue Oct 24 23:08:37 2023
    [email protected] <[email protected]> wrote:

    [snip]

    Point taken. But Behe is standing on the shoulders of giants
    for his dating of when "Sickle Trait Eve" was born, and it's
    quite important as to being able to show that the malaria parasite
    has had all this time to mutate a defense, and it has failed to do so.

    Is there consensus on a singular sickle trait eve? Seems it could have
    evolved independently several times.

    There’s this: “Since Allison and Haldane's work, the action of natural selection on genetic resistance to malaria has been shown in a multitude of contexts (Kwiatkowski, 2005). Indeed, the sickle-cell variant (i.e., the
    HbS allele) has been identified in four distinct genetic backgrounds in different African populations, suggesting that the same mutation arose independently several times through convergent evolution. Beyond HbS, other distinct mutations in the HBB gene have generated the HbC and HbE alleles, which arose and spread in Africa and in Southeast Asia, respectively”
    From: https://www.nature.com/scitable/topicpage/natural-selection-uncovering-mechanisms-of-evolutionary-adaptation-34539/

    Next up: “The sickle cell trait is widespread throughout Africa with low frequencies (<1%–2%) in the north and south of the continent and high but variable frequencies throughout much of equatorial Africa. Characterization
    of the DNA structure flanking the β-globin locus of HbS suggests that the mutation has arisen on at least three independent occasions in the African continent, referred to as β-globin haplotypes and named after the areas
    where they were first described: Benin, Senegal, and Central African
    Republic or Bantu (Pagnier et al. 1984; Nagel et al 1985; Chebloune et al. 1988). The HbC trait is believed to be a relatively recent mutation limited
    to West Africa where it occurs at high frequencies (>20%) in central Ghana
    and Burkina Faso, in only 2% in Nigeria, and does not occur, except in
    peoples of West African origin, in East and Central Africa. Only limited
    data are available on the type and distribution of α- and β-thalassemia
    genes in the African continent.”*
    From: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3784812/

    But there’s this: “There are two models of the origins of the sickle allele. The multicentric model posits five independent occurrences of the
    same mutation within the last few thousand years. The unicentric model
    posits a single occurrence and an older age. We used whole-genome-sequence
    data to provide insight into this issue. Using haplotypic classification
    and phylogenetic network analysis, we found clear and consistent evidence
    for a single origin of the sickle mutation. After accounting for
    recombination, we estimated that the sickle mutation is 259 [123,395] generations old.”
    From: https://www.cell.com/ajhg/fulltext/S0002-9297(18)30048-X

    This last article was referenced in https://www.bbc.com/news/world-africa-43373247.amp

    Where is was said: “In a study published on Thursday in the American
    Journal of Human Genetics, Daniel Shriner and Charles Rotimi from the
    Center for Research on Genomics and Global Health presented findings from analysing the genomes of nearly 3,000 people, 156 of whom had sickle cell.
    The researchers say they traced the mutation back for 7,300 years, and
    found it started with just one child.”
    […]
    “But Frederick B. Piel of Imperial College London has told the New York
    Times that he wants to see bigger studies to see if they come to the same conclusions.”

    “For decades scientists have wondered whether the mutation happened just once, or whether it happened at different times in different places.
    Sickle cells were first found in the US in people of African origin, but
    they are also common in people from the eastern Mediterranean (particularly Greece), the Middle East and parts of Asia.”
    […]
    “So is Dr Rotimi certain of what his study has found?
    At that question, he laughs out loud: "As a scientist it's always a bad
    idea to say something is final. I never really take the position that this
    is the final answer."”

    “But, he says: "The information that we have now seems to make it quite
    clear that the multiple origin is not supported."”

    So I dunno if it’s multicentric or unicentric in origin. Do you?

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Wed Oct 25 04:01:11 2023
    On Tue, 24 Oct 2023 14:30:19 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    This has been another super-busy day, so I'm posting only in reply
    to posts with comments that are very much on-topic for the purposes
    for which talk.origins and sci.bio.evolution were set up, and reciprocating.

    And since s.b.e. went extinct over a decade ago, its on-topic
    themes have been picked up by talk.origins and sci.bio.paleontology.

    On Sunday, October 22, 2023 at 3:31:15?AM UTC-4, jillery wrote:
    On Fri, 20 Oct 2023 17:25:27 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    On Wednesday, October 18, 2023 at 3:21:12?AM UTC-4, Burkhard wrote:

    Humanities-type ID research on gene-breakages should form (testable)
    hypothesis of the type:
    [...]
    The designer chose this method of gene breakages because...

    No, gene breakages are a purely Darwinian means of producing whole new
    species. Behe made that point right in the article I linked for jillery: >> >
    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Incorrect. Behe made no such claim.

    Not that you deserve it, but I'll give you the benefit of the doubt,
    and assume that you did not read as far as the last paragraph.
    Note the word "devolution" which Behe uses to mean "evolution that results from
    degradation of the genome."

    "At least in retrospect, it’s easy to see that devolution must happen — for the simple reason that helpful degradative mutations are more plentiful than helpful constructive ones and thus arrive more quickly for natural selection to multiply. The
    more recent results recounted here just pile more evidence onto that gathered in _Darwin_Devolves_ showing Darwin’s mechanism is powerfully devolutionary. That simple realization neatly explains results ranging from the evolutionary behavior of yeast
    in a comfy modern laboratory, to the speciation of megafauna in raw nature millions of years ago, and almost certainly to everything in between."

    Note the word "speciation" in that last half sentence.

    What Behe claims above is that
    gene breakage is "the most likely way" organisms evolve, and so "can’t
    account for the origins of sophisticated biological systems."

    Those are on the level of whole new families or orders,
    but Behe stopped with speciation above.


    He said nothing about creating whole new species,

    i.e., speciation -- but he did talk about that "loud and clear,"
    as the quoted comments show.


    I see that now. I have no excuse for missing it before. Now I have
    to rethink my understanding of what Behe's point is wrt devolution.
    Thank you for pointing out my mistake.


    or that gene breakage is
    limited to Darwinian means. In fact, gene breakage is entirely
    consistent with ID as well.

    Yes, but Burkhard was talking about divining the motivations of
    (possibly human-level) designers, and in the case of the breakages Behe is talking about,
    the Laplacian formula applies, "Sire, I had no need of that hypothesis."


    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Burkhard@21:1/5 to [email protected] on Wed Oct 25 05:15:15 2023
    On Wednesday, October 25, 2023 at 12:26:18 AM UTC+2, [email protected] wrote:
    On Saturday, October 21, 2023 at 5:31:15 PM UTC-4, Burkhard wrote:
    On Saturday, October 21, 2023 at 2:26:14 AM UTC+2, [email protected] wrote:
    On Wednesday, October 18, 2023 at 3:21:12 AM UTC-4, Burkhard wrote:
    On Wednesday, October 18, 2023 at 12:21:11 AM UTC+2, [email protected] wrote:
    On Saturday, October 14, 2023 at 2:21:08 PM UTC-4, Burkhard wrote:
    On Friday, October 13, 2023 at 2:36:06 AM UTC+2, [email protected] wrote:
    On Thursday, October 12, 2023 at 12:46:05 AM UTC-4, jillery wrote:
    On Wed, 11 Oct 2023 17:47:45 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    [two lines repeated from first reply, for context]
    Finally, whatever else Daggett might do, he does not make a career of
    posting false and misleading claims against evolution.

    Except, of course, out of ignorance. He thinks it is axiomatic that one's
    siblings are usually one's biggest competitors for survival. It's hard to guess
    where he picked up that "axiom", so instead of speculating,
    I reminded him of some "selfish gene" type folklore which he has to reconcile with that "axiom".

    [Oh, for the good old days of sci.bio.evolution, where genetics heavyweight
    Felsenstein put in regular appearances in the 1990's. That group went belly-up
    a decade ago due to boredom.]


    The next comment refers to the ignorant "chez watt" that claimed that
    IDers don't try to research the causes of speciation. Perhaps literally, yes,
    but if one expands "research" to what passes for that [1] in the humanities,
    then there is ample evidence ripe for the picking from scientific sources.

    Really, where?

    Do you understand what I wrote below about devolution by gene breakage?
    If so, you have one answer: Behe researches (in the humanities sense of combing existing documents)
    speciation that includes enough gene breakages to cause not only speciation, but
    giving rise to separate genera.

    Which humanities method is used in that research?

    Trying to find all relevant documents, and to study them in depth,
    and to weave what one has studied into a coherent hypothesis.

    That is not distinctive for humanities, that is just research synthesis, and is something that all disciplines do do - hence "standing on the shoulders of giants". Of course researchers don't have to do all experiments on their own, they also read the results of others, and try to build
    theories that account for all of them.
    That is exactly what Behe does with malaria in _The Edge of Evolution_, as I had written below.
    Gene breakage is a much newer topic for him, so not on the same high level yet,
    but his approach is promising.

    So with other words what he does is straightforward natural sciences research?
    This is all very confusing. You claim on the one hand that his research is more
    akin to the humanities, yet here you agree, again, with a characterisation of his research that is normal practice in the natural sciences?


    And in the natural sciences, meta-analysis is a rigorous research method, with
    the Cochrane review setting the gold standard.

    Behe did this to a much greater extent in _The Edge of Evolution_. He used malaria
    as an example of "trench warfare" between two antagonists [the malaria parasite and the human host]
    where lasting victory seems to be a matter of having at least three different mutations
    in the parasite, [with us humans it is different -- see below] all of which are beneficial
    under the circumstances dictated by the environment.

    I can't see any research methodology that is distinctive for the humanities there,
    this is just totally normal theory formation in the natural sciences.
    Yes, but I've been making the point that the research Behe does IS real research
    if we use the standards of the humanities for the word "research."

    This makes no sense at all, The part after the "yes" directly contradicts
    the "yes". Nothing you have said or shown here gives any indication whatsoever that Behe is doing research in the way that is distinctive
    for the humanities. And you explicitly agree that what he does is
    common for natural sciences. So how can you then argue that what
    he does is real research when evaluated against the understanding of
    research in the humanities?


    With the sickle cell trait -- NOT the disease [1] -- the parasite has been stymied; in contrast,
    one drug treatment after another has been successfully countered by it.

    I wonder whether Bill Rogers, our malaria professional, can either (1) find flaws with it or
    (2) has ever composed a more masterly account than the mere amateur, Michael Behe, has done.

    [1] The sickle cell trait is characterized as having one normal gene
    and one mutated version of it that protects against malaria.
    The disease only has the mutated version. It protects against malaria even a tad better,
    but it results in a debilitating anemia that is even worse than most cases of malaria.

    I can't see any.

    I haven't provided any until now. This is an ongoing discussion in which I don't
    try to anticipate your every criticism.


    And what documents do you mean? If you simply mean that rather
    than doing his own experiments,

    Just what "experiments" is someone trying to discover the author of
    the Voinich manuscript supposed to do?

    First, that misunderstands the argument entirely. Or are you really arguing that if A is lacking a trait X, and B is lacking the same trait X, A is B?
    Obviously not! Behe looks at numerous measures and counter-measures
    in this never-ending "trench warfare." There are numerous types of
    traits evolving on either side.

    I'm not talking about evolved traits, I'm talking about traits, or aspects,
    of his research.

    That is rather obviously fallacious. Footballers don't use javelins,
    and chess players don't use javelins, but that does not mean that
    football is the same as chess.
    Why do you make these analogies in the absence of any palpable
    statement by me to which they could be applicable?

    I'd say they are directly applicable. The only commonality you have
    stated between what Behe does and what people in the humanities do
    is "not to carry out experiments". That is, the commonality is only
    something they don't do. By the same logic, you'd have to conclude
    that footballers do sport by the criteria of chess, because both
    football and chess don't use javelins


    Even if it were true that humanities researchers do not carry out experiments,
    the fact that Behe also does not carry out experiments would not make him a humanities researcher, not by a long mile.
    Obviously not! I gave the main point I was making above:
    "Yes, but I've been making the point..." etc.

    And it still does not make sense. You seem to be arguing that he is both
    doing and not doing research the way humanities understand this

    But apart from being logically flawed, it's also wrong as a matter of fact. Of course
    Voynich researchers have and are carrying our experiments.Gregory Hodgins e.g.
    carried out a carbon dating analysis, that rules out several theories that treated
    it as a modern fake.
    Point taken. But Behe is standing on the shoulders of giants
    for his dating of when "Sickle Trait Eve" was born, and it's
    quite important as to being able to show that the malaria parasite
    has had all this time to mutate a defense, and it has failed to do so.
    The ink has equally been subjected to testing, as were the
    paint in the colourings (Barabe, Joseph G. (1 April 2009).
    "Materials Analysis of the Voynich Manuscript") - showing e.g. that text and images
    are contemporaneous, and that the author had access to some more
    unusual materials.


    or someone trying to figure
    out the hour in which the climactic Norman assault at Hastings took place?
    Let me know when you figure out an experiment that would
    shed light on this at this late date.

    Well, as R. Allen Brown put it, the only undisputed thing about the battle of Hastings
    is that the Norman's won. I don't know of any discussion about the timing, and on
    its own it would not be an interesting research question - for this it would need a
    theory where it plays some explanatory role, for instance "the 1. attack failed because
    the early morning hour meant the archers were blinded by the first light" or something
    like this (and somewhat trivially, you can check, or "test", where the sun stands at Hastings at
    various time of the day)

    As a bare fact, well the humanities approach is to look at the sources - there are no direct eyewitness
    accounts from Hastings, but seven independent contemporaneous sources that claim to
    have spoken to such witnesses (well, one of them only implicitly, being a poem and what not).
    These sources would then get interrogated by the normal canon: were they in a position to know,
    had they reasons to be distort the account etc. As in this case, those sources that mention
    the time at all all agree, the account fits to what we know from military doctrine at the time,
    and nothing depends on it anyway, you'll find provisional timings in the literature (essentially from
    dusk til dawn, i.e. starting probably around 9 am).

    But I still don't get what this has to do with Behe. Does he have eyewitness statements from
    the design event, or failing this sources who spoke to these eyewitnesses, and did he analyse
    them as to their epistemological quality? Does what he suggests "fit" what we know about
    the way designers normally build things such as flagella? I don't see any of this myself.

    he uses those made by other, sorry,
    no, mere "displaying reading ability" does not make this into humanities-type
    research.

    See what I wrote about Behe and malaria. He's gone into lots of angles that one does
    not find in standard treatments of the subject, or in the writings of any single author.
    Certainly not in the writings of Bill Rogers.

    And that turns it into humanities research how?
    Missing the main point again.
    Humanities-type ID research on gene-breakages should form (testable) hypothesis of the type: The designer wanted these genera because....
    A creationist would frame such hypotheses, but Behe allows for lots of unguided evolution.


    But he also argues design, and a design theory would have to use
    these hypothesis somewhere.

    Yes...in very carefully chosen cases that call for it. A good design theorist does not go hunting for design under every bed, as the metaphor goes.

    That's fine, nobody asks him to. So where are the case studies where he develops
    testable hypothesis about motives, means and opportunities of the designer?

    So his hypotheses are of a different sort, like the
    one I mentioned above: how likely is that unguided processes will
    arrive at a successful counter-measure of the parasite to the entrapment and suffocation the sickle cell trait "uses" to kill the parasites?


    That quantifies over all processes, including unknown ones.
    That is why it is so important that the sickle cell trait has been
    around for thousands of years. In contrast, the usual drug based
    treatments get mutated around every few years, because of
    the staggering number of individual parasites in every infected person.
    That is
    just bad research methodology.
    No worse than quantifying over all the unknown people that
    produced the ancient Indus Valley script. Despite about
    a century and a half of effort, no one has deciphered a single sentence.

    Erm, no? That analogy does not work for you at all, quite on the contrary. You'd have to conclude that the absence of a detailed mechanism of
    how the Harappan script evolved from earlier scripts (and the language
    from earlier languages), and we don't know how it is related to any
    other known script, we should conclude that it could not come
    from any of the cultures in the Indus Valley, and instead was intelligently designed by an unknown, probably non-human, designer. But of course
    no serious researcher argues that - precisely because that type
    of quantification (no possible.... coule have...) is unsound.

    Doing natural sciences badly does not
    mean doing it like the humanities do

    A vast period of time involving several million years might overcome the odds,
    but neither humans nor the parasite have been around that long.
    Humans, on the other hand, needed only ONE mutation in ONE human with several offspring to produce the sickle cell trait. Consequently, Behe doesn't claim
    that the mutation was divinely caused or even that the Designer foresaw it.

    So why then is it a design theory?
    The theory has to do with how many successive neutral or slightly deleterious
    mutations it takes to evolve a very precise certain feature, before "design" is hypothesized. I'm rushed for time, but IIRC Behe puts "the edge of evolution" at "more than three."


    <snip for focus>
    or The designer chose this method of gene breakages because...

    No, gene breakages are a purely Darwinian means of producing whole new species. Behe made that point right in the article I linked for jillery:

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/
    And this is a really strange argument for you to make.

    Just playing in your ballpark. [Do you recognize the allusion to a key juncture in the film,
    "Inherit the Wind"?]

    <crickets>

    nothing worth commenting on, really
    Then you've lost the "strange argument" game on your home turf. Would you like a rematch?


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of So. Carolina at Columbia
    http://people.math.sc.edu/nyikos

    PS you had nothing new to say below, but I am pressed for time and haven't commented on what's there either. But I left it in below, intact.
    You didn't say anything more this time around, but I've left in some of what you left in,
    with one comment about what you did say this time around.
    I have said
    numerous times that IDlers do NOT develop theories about their subject matter the
    way the humanities do - only for you to claim that because they are natural scientists
    (ha-ha) that would be an inappropriate demand on them .

    That was before I realized how the theories apply scientific understanding to the results of
    research methods employed in the humanities.
    <snip for focus>
    The following is an instance of Behe's "evolution by devolution"":

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    The woolly mammoth is *Mammuthus* *primigenius,* a separate genus
    from the African elephant (Loxodonta) and the Asian elephant (Elephas).[2]
    Behe is quite comfortable with the idea that the great number of genes
    that were broken were adequate to launch a new species or even a new genus.

    [2] Elephas is the closest living relative of the woolly mammoth. This is what I was alluding to up there, this time around.
    <small snip>
    This was a result of Behe researching some of the latest scientific news:

    The following research announcement in Science News from last year talks
    about those broken genes.

    https://www.sciencedaily.com/releases/2022/08/220810123606.htm "Nearly a hundred genes have been lost during the woolly mammoth's evolution"
    Date: August 10, 2022
    <snip for focus>

    This article was reported five days later by Michael Behe and
    conclusions were drawn from its findings.

    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    If it has flaws, you should be grateful that I talked about it here:
    it would keep you from being blindsided later on.

    If you can't find any flaws with it, it would give you plenty of time
    to spin-doctor its conclusions in a way that cushions the shock of it being quoted to you.

    A win-win situation, if you can bring yourself to read it.

    Jillery hasn't responded yet. Do you think you could help her out by spotting some flaws -- like,
    telling us how such simple "research" doesn't do justice to the way people in the humanities do research?
    You made a weak stab at it this time, but you may add more in the light of what I wrote this time.
    Such back-and-forth is what Usenet was designed for.
    Peter Nyikos
    Professor, Dept. of Mathematics
    U. of So. Carolina -- standard disclaimer-- https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From *Hemidactylus*@21:1/5 to [email protected] on Thu Oct 26 10:05:34 2023
    *Hemidactylus* <[email protected]d> wrote:
    [email protected] <[email protected]> wrote:

    [snip]

    Point taken. But Behe is standing on the shoulders of giants
    for his dating of when "Sickle Trait Eve" was born, and it's
    quite important as to being able to show that the malaria parasite
    has had all this time to mutate a defense, and it has failed to do so.

    Is there consensus on a singular sickle trait eve? Seems it could have evolved independently several times.

    There’s this: “Since Allison and Haldane's work, the action of natural selection on genetic resistance to malaria has been shown in a multitude of contexts (Kwiatkowski, 2005). Indeed, the sickle-cell variant (i.e., the
    HbS allele) has been identified in four distinct genetic backgrounds in different African populations, suggesting that the same mutation arose independently several times through convergent evolution. Beyond HbS, other distinct mutations in the HBB gene have generated the HbC and HbE alleles, which arose and spread in Africa and in Southeast Asia, respectively”
    From: https://www.nature.com/scitable/topicpage/natural-selection-uncovering-mechanisms-of-evolutionary-adaptation-34539/

    Next up: “The sickle cell trait is widespread throughout Africa with low frequencies (<1%–2%) in the north and south of the continent and high but variable frequencies throughout much of equatorial Africa. Characterization of the DNA structure flanking the β-globin locus of HbS suggests that the mutation has arisen on at least three independent occasions in the African continent, referred to as β-globin haplotypes and named after the areas where they were first described: Benin, Senegal, and Central African
    Republic or Bantu (Pagnier et al. 1984; Nagel et al 1985; Chebloune et al. 1988). The HbC trait is believed to be a relatively recent mutation limited to West Africa where it occurs at high frequencies (>20%) in central Ghana and Burkina Faso, in only 2% in Nigeria, and does not occur, except in peoples of West African origin, in East and Central Africa. Only limited
    data are available on the type and distribution of α- and β-thalassemia genes in the African continent.”*
    From: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3784812/

    But there’s this: “There are two models of the origins of the sickle allele. The multicentric model posits five independent occurrences of the same mutation within the last few thousand years. The unicentric model
    posits a single occurrence and an older age. We used whole-genome-sequence data to provide insight into this issue. Using haplotypic classification
    and phylogenetic network analysis, we found clear and consistent evidence
    for a single origin of the sickle mutation. After accounting for recombination, we estimated that the sickle mutation is 259 [123,395] generations old.”
    From: https://www.cell.com/ajhg/fulltext/S0002-9297(18)30048-X

    This last article was referenced in https://www.bbc.com/news/world-africa-43373247.amp

    Where is was said: “In a study published on Thursday in the American Journal of Human Genetics, Daniel Shriner and Charles Rotimi from the
    Center for Research on Genomics and Global Health presented findings from analysing the genomes of nearly 3,000 people, 156 of whom had sickle cell. The researchers say they traced the mutation back for 7,300 years, and
    found it started with just one child.”
    […]
    “But Frederick B. Piel of Imperial College London has told the New York Times that he wants to see bigger studies to see if they come to the same conclusions.”

    “For decades scientists have wondered whether the mutation happened just once, or whether it happened at different times in different places.
    Sickle cells were first found in the US in people of African origin, but
    they are also common in people from the eastern Mediterranean (particularly Greece), the Middle East and parts of Asia.”
    […]
    “So is Dr Rotimi certain of what his study has found?
    At that question, he laughs out loud: "As a scientist it's always a bad
    idea to say something is final. I never really take the position that this
    is the final answer."”

    “But, he says: "The information that we have now seems to make it quite clear that the multiple origin is not supported."”

    So I dunno if it’s multicentric or unicentric in origin. Do you?

    Crickets chirping in the gentle topical breeze.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Thu Oct 26 12:14:50 2023
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.


    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Thu Oct 26 12:51:08 2023
    On Wednesday, October 25, 2023 at 4:06:19 AM UTC-4, jillery wrote:
    On Tue, 24 Oct 2023 14:30:19 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    This has been another super-busy day, so I'm posting only in reply
    to posts with comments that are very much on-topic for the purposes
    for which talk.origins and sci.bio.evolution were set up, and reciprocating.

    And since s.b.e. went extinct over a decade ago, its on-topic
    themes have been picked up by talk.origins and sci.bio.paleontology.

    On Sunday, October 22, 2023 at 3:31:15?AM UTC-4, jillery wrote:
    On Fri, 20 Oct 2023 17:25:27 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    On Wednesday, October 18, 2023 at 3:21:12?AM UTC-4, Burkhard wrote:

    Humanities-type ID research on gene-breakages should form (testable) >>>> hypothesis of the type:
    [...]
    The designer chose this method of gene breakages because...

    No, gene breakages are a purely Darwinian means of producing whole new >> >species. Behe made that point right in the article I linked for jillery: >> >
    https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Incorrect. Behe made no such claim.

    Not that you deserve it, but I'll give you the benefit of the doubt,
    and assume that you did not read as far as the last paragraph.
    Note the word "devolution" which Behe uses to mean "evolution that results from
    degradation of the genome."

    "At least in retrospect, it’s easy to see that devolution must happen — for the simple reason that helpful degradative mutations are more plentiful than helpful constructive ones and thus arrive more quickly for natural selection to multiply. The
    more recent results recounted here just pile more evidence onto that gathered in _Darwin_Devolves_ showing Darwin’s mechanism is powerfully devolutionary. That simple realization neatly explains results ranging from the evolutionary behavior of yeast
    in a comfy modern laboratory, to the speciation of megafauna in raw nature millions of years ago, and almost certainly to everything in between."

    Note the word "speciation" in that last half sentence.

    What Behe claims above is that
    gene breakage is "the most likely way" organisms evolve, and so "can’t >> account for the origins of sophisticated biological systems."

    Those are on the level of whole new families or orders,
    but Behe stopped with speciation above.


    He said nothing about creating whole new species,

    i.e., speciation -- but he did talk about that "loud and clear,"
    as the quoted comments show.-+
    I see that now. I have no excuse for missing it before. Now I have
    to rethink my understanding of what Behe's point is wrt devolution.
    Thank you for pointing out my mistake.

    Thank you for this most gracious acknowledgement. It makes me glad that
    I gave you the benefit of the doubt. In hindsight, I could have done a better job of that. But all's well that ends well.

    or that gene breakage is
    limited to Darwinian means. In fact, gene breakage is entirely
    consistent with ID as well.

    Yes, but Burkhard was talking about divining the motivations of
    (possibly human-level) designers, and in the case of the breakages Behe is talking about,
    the Laplacian formula applies, "Sire, I had no need of that hypothesis."

    Behe would probably put it a little differently, perhaps ending with something like,

    "Give unto science the things that belong to science, and unto the designer[s] [1]
    the things that seem to be [2] due to the designer[s]."

    Devout Catholic that he is, he might privately think "God," when writing " the designer[s]." But that
    would no longer be thinking *qua* ID theorist, but *qua* devout Catholic.

    [1] Note the lower case, to include non-supernatural designers, including some of no greater
    level of intelligence than ourselves.

    [2] With a big gray area of suspended judgment, fluctuating as new data comes in.


    Peter Nyikos
    Professor, Dept. of Mathematics
    University of So. Carolina -- standard disclaimer-- http://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to All on Thu Oct 26 15:51:19 2023
    On Thursday, October 26, 2023 at 6:06:20 AM UTC-4, *Hemidactylus* wrote:
    *Hemidactylus* <[email protected]d> wrote:
    [email protected] <[email protected]> wrote:

    [snip]

    Point taken. But Behe is standing on the shoulders of giants
    for his dating of when "Sickle Trait Eve" was born, and it's
    quite important as to being able to show that the malaria parasite
    has had all this time to mutate a defense, and it has failed to do so.

    Is there consensus on a singular sickle trait eve? Seems it could have evolved independently several times.

    There’s this: “Since Allison and Haldane's work, the action of natural selection on genetic resistance to malaria has been shown in a multitude of
    contexts (Kwiatkowski, 2005). Indeed, the sickle-cell variant (i.e., the HbS allele) has been identified in four distinct genetic backgrounds in different African populations, suggesting that the same mutation arose independently several times through convergent evolution. Beyond HbS, other
    distinct mutations in the HBB gene have generated the HbC and HbE alleles, which arose and spread in Africa and in Southeast Asia, respectively” From: https://www.nature.com/scitable/topicpage/natural-selection-uncovering-mechanisms-of-evolutionary-adaptation-34539/

    Next up: “The sickle cell trait is widespread throughout Africa with low frequencies (<1%–2%) in the north and south of the continent and high but
    variable frequencies throughout much of equatorial Africa. Characterization
    of the DNA structure flanking the β-globin locus of HbS suggests that the mutation has arisen on at least three independent occasions in the African continent, referred to as β-globin haplotypes and named after the areas where they were first described: Benin, Senegal, and Central African Republic or Bantu (Pagnier et al. 1984; Nagel et al 1985; Chebloune et al. 1988). The HbC trait is believed to be a relatively recent mutation limited
    to West Africa where it occurs at high frequencies (>20%) in central Ghana and Burkina Faso, in only 2% in Nigeria, and does not occur, except in peoples of West African origin, in East and Central Africa. Only limited data are available on the type and distribution of α- and β-thalassemia genes in the African continent.”*
    From: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3784812/

    But there’s this: “There are two models of the origins of the sickle allele. The multicentric model posits five independent occurrences of the same mutation within the last few thousand years. The unicentric model posits a single occurrence and an older age. We used whole-genome-sequence data to provide insight into this issue. Using haplotypic classification and phylogenetic network analysis, we found clear and consistent evidence for a single origin of the sickle mutation. After accounting for recombination, we estimated that the sickle mutation is 259 [123,395] generations old.”
    From: https://www.cell.com/ajhg/fulltext/S0002-9297(18)30048-X

    This last article was referenced in https://www.bbc.com/news/world-africa-43373247.amp

    Thanks for including this. The original is very technical and I don't know
    when I'll have time for looking it over. This easy-reading report has the following key claim:

    "But Dr Shriner and Dr Rotimi found that the people they traced had very similar genetic mutations, with those in Kenya, Uganda, South Africa and the Central African Republic being so similar that they would fit a pattern of having been distributed by
    migrations of the Bantu people.

    "The Bantu, from West Africa, moved eastward and southward about 2,500 years ago.
    [end of quote]

    Perhaps, they interbred with people of very different background. And some of these
    may have migrated far away, accounting for the other locales.

    A lot can happen in 2,500 years. It's possible that the mini-continent of Madagascar
    had never had a human on it back then. Six greatly varied kinds of lemurs, all bigger than
    the largest ones alive today; the elephant bird (possibly the heaviest bird that ever lived)
    and other now-extinct animals, may still have been walking around.


    Where is was said: “In a study published on Thursday in the American Journal of Human Genetics, Daniel Shriner and Charles Rotimi from the Center for Research on Genomics and Global Health presented findings from analysing the genomes of nearly 3,000 people, 156 of whom had sickle cell. The researchers say they traced the mutation back for 7,300 years, and found it started with just one child.”
    […]
    “But Frederick B. Piel of Imperial College London has told the New York Times that he wants to see bigger studies to see if they come to the same conclusions.”

    “For decades scientists have wondered whether the mutation happened just once, or whether it happened at different times in different places. Sickle cells were first found in the US in people of African origin, but they are also common in people from the eastern Mediterranean (particularly
    Greece), the Middle East and parts of Asia.”
    […]
    “So is Dr Rotimi certain of what his study has found?
    At that question, he laughs out loud: "As a scientist it's always a bad idea to say something is final. I never really take the position that this is the final answer."”

    “But, he says: "The information that we have now seems to make it quite clear that the multiple origin is not supported."”

    So I dunno if it’s multicentric or unicentric in origin. Do you?

    No. A lot of what you dug up is new to me, and will take a long time to digest.


    Crickets chirping in the gentle topical breeze.

    Gimme a break, Hemi. This week has been so busy with unexpected
    things here, both at work and impacting my family, that I've been
    alternating between threads from day to day, each with a few
    carefully chosen posts. And sci.bio.paleontology hasn't had a post
    from me this week at all yet.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    Univ. of South Carolina at Columbia
    http://people.math.sc.edu/nyikos

    PS Thanks for doing so much legwork. I'll be looking more closely
    at your references this weekend.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Fri Oct 27 12:16:20 2023
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus* <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the degradation of the genome.


    Peter Nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From *Hemidactylus*@21:1/5 to [email protected] on Fri Oct 27 19:37:33 2023
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside
    from being an implicit value judgment?

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to All on Fri Oct 27 14:25:42 2023
    On Friday, October 27, 2023 at 3:41:21 PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism, the clearest form is when a nucleotide is replaced by another one in such
    a way that the triplet to which it belongs still codes for the same amino acid. Is that what you were referring to as the predominate mode?

    That is incompatible with the breaking of a gene, which not only prevents it from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the events that transformed an elephant into a mammoth, that adapted the animal to its changing environment. A job well done, yes, but now those genes are gone forever, unavailable to
    help with the next change of environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Jillery took the words "gone forever" literally, but with the extinction of the woolly mammoth,
    it's a moot point. There are some who hope to resurrect mammoths by cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because the germ cell into which the nucleus of a mammoth cell is placed has an effect on which genes
    are expressed. The embryonic development is influenced by the mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical mammoth on our hands.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of South Carolina at Columbia
    http://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Sat Oct 28 00:45:06 2023
    On Fri, 27 Oct 2023 14:25:42 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Friday, October 27, 2023 at 3:41:21?PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20?PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside >> from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such
    a way that the triplet to which it belongs still codes for the same amino acid.
    Is that what you were referring to as the predominate mode?

    That is incompatible with the breaking of a gene, which not only prevents it >from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the events that transformed an elephant into a mammoth, that adapted the animal to its changing environment. A job well done, yes, but now those genes are gone forever, unavailable to
    help with the next change of environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Jillery took the words "gone forever" literally,


    To be accurate, Behe dishonestly exaggerated the facts.


    but with the extinction of the woolly mammoth, it's a moot point.


    If so, then mammoth extinction would also moot Behe's entire line of
    reasoning.


    There are some who hope to resurrect mammoths by cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because >the germ cell into which the nucleus of a mammoth cell is placed has an effect on which genes
    are expressed. The embryonic development is influenced by the mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical mammoth on our hands.


    What you allude to above is a point I made previously about Dolly the
    sheep "clone" which was more accurately a chimera.

    Another problem with resurrecting extinct species is there are no
    parents to teach these organisms how to act appropriately. For these
    reasons and others, these experiments produce approximate models of
    extinct species.


    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From *Hemidactylus*@21:1/5 to [email protected] on Sat Oct 28 07:26:20 2023
    [email protected] <[email protected]> wrote:
    On Friday, October 27, 2023 at 3:41:21 PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside >> from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism, the clearest form is when a nucleotide is replaced by another one in such
    a way that the triplet to which it belongs still codes for the same amino acid.
    Is that what you were referring to as the predominate mode?

    Or a duplicate gene degrades while the original still remains functional.
    Or retroviruses and transposons litter the genome and remain as fossils.
    There are multiple ways junk accumulates. The point mutational view of evolution is a bit blinkered.

    Devolution conjures the dichotomy of the Morlocks and Eloi reflecting a
    turn of the century obsession with degeneration.

    https://www.merriam-webster.com/dictionary/devolve

    “…to degenerate through a gradual change or evolution”

    Reminds me of popular fears of inbreds that inspire horror movies like
    “Wrong Turn”.

    Degradation of genes from an adaptive state such as with our remnant
    yolking genes is not devolution but merely evolution. Are we devolved from
    our egg-laying ancestors because we don’t yolk our eggs?

    That is incompatible with the breaking of a gene, which not only prevents it from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the events that transformed an elephant into a mammoth, that adapted the
    animal to its changing environment. A job well done, yes, but now those
    genes are gone forever, unavailable to help with the next change of environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Degradation of yolking genes in our ancestors should have done them in. Yet here we are with those broken genes. Amazing!

    Jillery took the words "gone forever" literally, but with the extinction
    of the woolly mammoth,
    it's a moot point. There are some who hope to resurrect mammoths by
    cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because
    the germ cell into which the nucleus of a mammoth cell is placed has an effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical mammoth on our hands.

    Ah yes epigenetics and the EES.

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Ernest Major@21:1/5 to All on Sun Oct 29 11:52:12 2023
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Friday, October 27, 2023 at 3:41:21 PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside >>> from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such
    a way that the triplet to which it belongs still codes for the same amino acid.
    Is that what you were referring to as the predominate mode?

    Or a duplicate gene degrades while the original still remains functional.
    Or retroviruses and transposons litter the genome and remain as fossils. There are multiple ways junk accumulates. The point mutational view of evolution is a bit blinkered.

    Devolution conjures the dichotomy of the Morlocks and Eloi reflecting a
    turn of the century obsession with degeneration.

    https://www.merriam-webster.com/dictionary/devolve

    “…to degenerate through a gradual change or evolution”

    Reminds me of popular fears of inbreds that inspire horror movies like “Wrong Turn”.

    Degradation of genes from an adaptive state such as with our remnant
    yolking genes is not devolution but merely evolution. Are we devolved from our egg-laying ancestors because we don’t yolk our eggs?

    That is incompatible with the breaking of a gene, which not only prevents it >> from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the
    events that transformed an elephant into a mammoth, that adapted the
    animal to its changing environment. A job well done, yes, but now those
    genes are gone forever, unavailable to help with the next change of
    environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Degradation of yolking genes in our ancestors should have done them in. Yet here we are with those broken genes. Amazing!

    Jillery took the words "gone forever" literally, but with the extinction
    of the woolly mammoth,
    it's a moot point. There are some who hope to resurrect mammoths by
    cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because
    the germ cell into which the nucleus of a mammoth cell is placed has an
    effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical
    mammoth on our hands.

    Ah yes epigenetics and the EES.


    The interaction between the mitome and nucleome isn't epigenetics. The
    term that applies to traits that result from the interaction of
    different genes is polygenic.

    There's also the point that if one could assemble a mammoth nucleome one
    could assemble a mammoth mitome. The first step in resurrecting mammoths
    might even to be create an elephant with mammoth mitochondria.

    There does remain questions about the interaction between the uterine environment and the genome. (Nurture, not epigenetics.) Cross-species
    surrogacy is a technique used in conservation, so there is some
    knowledge as to how significant this is.

    And there is the question of the interaction between the ovum proteome
    and the genome. Elephant and mammoth DNA binding proteins may bind to
    the mammoth genome with different avidities, with impacts on gene
    regulation. This is at least getting close to epigenetics.

    Genomes can produce very different results - sexual dimorphism (are
    there species with non-chromosomal sex determination and significant
    sexual dimorphism beyond the reproductive system?), social insect
    castes, et alia. It's not beyond the bounds of conceivability that the
    cloning process kicks mammoth development into a different
    generationally persistent (epigenetically determined) phenotypic
    attractor, but my expectation that any impacts from the last two issue
    above could be eliminated by a few rounds of mammoth reproduction,
    presuming that you could produce a viable mammoth in the first place.

    With current technology it strikes me as more feasible to modify
    elephant genomes step by step using CRISPR and hope that the
    intermediates selected are viable than to recreate mammoths in a single generation. I suspect that we're closer to directed abiogenesis that to
    the latter.

    --
    alias Ernest Major

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From John Harshman@21:1/5 to Ernest Major on Sun Oct 29 06:50:50 2023
    On 10/29/23 4:52 AM, Ernest Major wrote:
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Friday, October 27, 2023 at 3:41:21 PM UTC-4, *Hemidactylus* wrote: >>>> [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote: >>>>>> On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution
    aside
    from being an implicit value judgment?

      If by "neutral evolution" you mean no effect on the fitness of the
    organism,
    the clearest form is when a nucleotide is replaced by another one in
    such
    a way that the triplet to which it belongs still codes for the same
    amino acid.
    Is that what you were referring to as the predominate mode?

    Or a duplicate gene degrades while the original still remains functional.
    Or retroviruses and transposons litter the genome and remain as fossils.
    There are multiple ways junk accumulates. The point mutational view of
    evolution is a bit blinkered.

    Devolution conjures the dichotomy of the Morlocks and Eloi reflecting a
    turn of the century obsession with degeneration.

    https://www.merriam-webster.com/dictionary/devolve

    “…to degenerate through a gradual change or evolution”

    Reminds me of popular fears of inbreds that inspire horror movies like
    “Wrong Turn”.

    Degradation of genes from an adaptive state such as with our remnant
    yolking genes is not devolution but merely evolution. Are we devolved
    from
    our egg-laying ancestors because we don’t yolk our eggs?

    That is incompatible with the breaking of a gene, which not only
    prevents it
    from coding for the same polypeptide, it even prevents it from coding
    for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the >>> events that transformed an elephant into a mammoth, that adapted the
    animal to its changing environment. A job well done, yes, but now those
    genes are gone forever, unavailable to help with the next change of
    environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Degradation of yolking genes in our ancestors should have done them
    in. Yet
    here we are with those broken genes. Amazing!

    Jillery took the words "gone forever" literally, but with the extinction >>> of the woolly mammoth,
    it's a moot point. There are some who hope to resurrect mammoths by
    cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success.
    That's because
    the germ cell into which the nucleus of a mammoth cell is placed has an
    effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical
    mammoth on our hands.

    Ah yes epigenetics and the EES.


    The interaction between the mitome and nucleome isn't epigenetics. The
    term that applies to traits that result from the interaction of
    different genes is polygenic.

    There's also the point that if one could assemble a mammoth nucleome one could assemble a mammoth mitome. The first step in resurrecting mammoths might even to be create an elephant with mammoth mitochondria.

    There does remain questions about the interaction between the uterine environment and the genome. (Nurture, not epigenetics.) Cross-species surrogacy is a technique used in conservation, so there is some
    knowledge as to how significant this is.

    And there is the question of the interaction between the ovum proteome
    and the genome. Elephant and mammoth DNA binding proteins may bind to
    the mammoth genome with different avidities, with impacts on gene
    regulation. This is at least getting close to epigenetics.

    There are also maternal transcripts, RNAs of various sorts.

    Genomes can produce very different results - sexual dimorphism (are
    there species with non-chromosomal sex determination and significant
    sexual dimorphism beyond the reproductive system?), social insect
    castes, et alia. It's not beyond the bounds of conceivability that the cloning process kicks mammoth development into a different
    generationally persistent (epigenetically determined) phenotypic
    attractor, but my expectation that any impacts from the last two issue
    above could be eliminated by a few rounds of mammoth reproduction,
    presuming that you could produce a viable mammoth in the first place.

    With current technology it strikes me as more feasible to modify
    elephant genomes step by step using CRISPR and hope that the
    intermediates selected are viable than to recreate mammoths in a single generation. I suspect that we're closer to directed abiogenesis that to
    the latter.


    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to Ernest Major on Tue Oct 31 12:03:58 2023
    On Sunday, October 29, 2023 at 7:56:23 AM UTC-4, Ernest Major wrote:
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:

    the germ cell into which the nucleus of a mammoth cell is placed has an >> effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical
    mammoth on our hands.

    Ah yes epigenetics and the EES.

    The interaction between the mitome and nucleome isn't epigenetics.

    Thanks for the correction.


    The term that applies to traits that result from the interaction of different genes is polygenic.

    Is there a term which applies specifically to the interaction
    between the genes of the nucleus and those of the mitochondria?

    There's also the point that if one could assemble a mammoth nucleome one could assemble a mammoth mitome. The first step in resurrecting mammoths might even to be create an elephant with mammoth mitochondria.

    There does remain questions about the interaction between the uterine environment and the genome. (Nurture, not epigenetics.) Cross-species surrogacy is a technique used in conservation, so there is some
    knowledge as to how significant this is.

    So, if by some miracle we could obtain a viable oocyte from a female
    mammoth, and viable sperm from a male, and get them to unite somehow, implanting the resulting blastocyst into an elephant could produce a
    genuine mammoth, no?

    And there is the question of the interaction between the ovum proteome
    and the genome. Elephant and mammoth DNA binding proteins may bind to
    the mammoth genome with different avidities, with impacts on gene regulation. This is at least getting close to epigenetics.

    Genomes can produce very different results - sexual dimorphism (are
    there species with non-chromosomal sex determination and significant
    sexual dimorphism beyond the reproductive system?), social insect
    castes, et alia. It's not beyond the bounds of conceivability that the cloning process kicks mammoth development into a different
    generationally persistent (epigenetically determined) phenotypic
    attractor, but my expectation that any impacts from the last two issue
    above could be eliminated by a few rounds of mammoth reproduction,
    presuming that you could produce a viable mammoth in the first place.

    With current technology it strikes me as more feasible to modify
    elephant genomes step by step using CRISPR and hope that the
    intermediates selected are viable than to recreate mammoths in a single generation. I suspect that we're closer to directed abiogenesis that to
    the latter.

    Directed abiogenesis might be something to shoot for, but with
    a simpler goal than "life as we know it."

    I've often wondered how diverse the earth biosphere could have become if the final stages of the "protein takeover" had not taken place, but stopped with ribozymes being the main catalysts rather than protein enzymes.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of South Carolina in Columbia
    https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Tue Oct 31 12:34:30 2023
    On Saturday, October 28, 2023 at 12:46:22 AM UTC-4, jillery wrote:
    On Fri, 27 Oct 2023 14:25:42 -0700 (PDT), "[email protected]" <[email protected]> wrote:
    On Friday, October 27, 2023 at 3:41:21?PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20?PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside
    from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such >a way that the triplet to which it belongs still codes for the same amino acid.
    Is that what you were referring to as the predominate mode?

    That is incompatible with the breaking of a gene, which not only prevents it
    from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the events that transformed an elephant into a mammoth, that adapted the animal to its changing environment. A job well done, yes, but now those genes are gone forever, unavailable
    to help with the next change of environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Jillery took the words "gone forever" literally,

    To be accurate, Behe dishonestly exaggerated the facts.

    Not so fast. There is more than one gene involved here.

    Excerpt from linked article:
    In Darwin Devolves, I also mentioned work on DNA extracted from frozen woolly mammoth carcasses that showcased devolution: “26 genes were shown to be seriously degraded, many of which (as with polar bear) were involved in fat metabolism, critical in
    the extremely cold environments that the mammoth roamed.”
    [end of excerpt]

    Even if one of those genes somehow got resurrected from its broken
    state to its original state -- unlikely in the time from the first woollymammoth
    to historical times [1]-- the probability of it happening to the majority
    of the relevant genes is so small as to make it a practical impossibility.

    [1] "once in a million blue moons" was the way I put it, and I don't think that's an exaggeration

    Moreover, junk DNA alters more quickly than the genes that are
    expressed, due to the effects of natural selection not applying to them.
    If one of the broken ones did come to be expressed again,
    it could be that it would no longer code for its original polypeptide,
    but for another one that is not useful for the purpose of suppressing
    the amount of insulating fat. This would put the individual at
    a disadvantage in times of global warming.

    but with the extinction of the woolly mammoth, it's a moot point.

    If so, then mammoth extinction would also moot Behe's entire line of reasoning.

    It was quite strong while the mammoth was in existence.
    And it still applies to polar bears, another species discussed
    by Behe in the linked article. Also to musk oxen and other cold-adapted mammals.


    There are some who hope to resurrect mammoths by cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because
    the germ cell into which the nucleus of a mammoth cell is placed has an effect on which genes
    are expressed. The embryonic development is influenced by the mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical mammoth on our hands.

    What you allude to above is a point I made previously about Dolly the
    sheep "clone" which was more accurately a chimera.

    Another problem with resurrecting extinct species is there are no
    parents to teach these organisms how to act appropriately. For these
    reasons and others, these experiments produce approximate models of
    extinct species.

    Good points.


    Peter Nyikos
    Professor, Dept. of Mathematics
    U. of South Carolina -- standard disclaimer-- https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Ernest Major@21:1/5 to [email protected] on Tue Oct 31 20:20:16 2023
    On 31/10/2023 19:03, [email protected] wrote:
    On Sunday, October 29, 2023 at 7:56:23 AM UTC-4, Ernest Major wrote:
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:

    the germ cell into which the nucleus of a mammoth cell is placed has an >>>> effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical >>>> mammoth on our hands.

    Ah yes epigenetics and the EES.

    The interaction between the mitome and nucleome isn't epigenetics.

    Thanks for the correction.


    The term that applies to traits that result from the interaction of
    different genes is polygenic.

    Is there a term which applies specifically to the interaction
    between the genes of the nucleus and those of the mitochondria?

    Not that I know of. (Cytoplasmic male sterility applies - assuming it's
    the mitochondria rather than the plastids that are involved - but that's
    only one of many possible interactions.)


    There's also the point that if one could assemble a mammoth nucleome one
    could assemble a mammoth mitome. The first step in resurrecting mammoths
    might even to be create an elephant with mammoth mitochondria.

    There does remain questions about the interaction between the uterine
    environment and the genome. (Nurture, not epigenetics.) Cross-species
    surrogacy is a technique used in conservation, so there is some
    knowledge as to how significant this is.

    So, if by some miracle we could obtain a viable oocyte from a female
    mammoth, and viable sperm from a male, and get them to unite somehow, implanting the resulting blastocyst into an elephant could produce a
    genuine mammoth, no?

    s/might/could/ - you might get a failed pregancy, or a genuine mammoth
    near as makes not difference, or something else. I know that there are experimental results for some species pairs, but I don't know the
    details of the results. The results of cloning by inserting a somatic
    cell nucleus, suitably reprogrammed, into a same species ovum might also
    shed light on how sensitive development is to perturbation.

    And there is the question of the interaction between the ovum proteome
    and the genome. Elephant and mammoth DNA binding proteins may bind to
    the mammoth genome with different avidities, with impacts on gene
    regulation. This is at least getting close to epigenetics.

    Genomes can produce very different results - sexual dimorphism (are
    there species with non-chromosomal sex determination and significant
    sexual dimorphism beyond the reproductive system?), social insect
    castes, et alia. It's not beyond the bounds of conceivability that the
    cloning process kicks mammoth development into a different
    generationally persistent (epigenetically determined) phenotypic
    attractor, but my expectation that any impacts from the last two issue
    above could be eliminated by a few rounds of mammoth reproduction,
    presuming that you could produce a viable mammoth in the first place.

    With current technology it strikes me as more feasible to modify
    elephant genomes step by step using CRISPR and hope that the
    intermediates selected are viable than to recreate mammoths in a single
    generation. I suspect that we're closer to directed abiogenesis that to
    the latter.

    Directed abiogenesis might be something to shoot for, but with
    a simpler goal than "life as we know it."

    It strikes me that assembling "life as we know it" (in the form of
    simple bacterium) is a much simpler problem than designing an organism
    from scratch.

    I don't know that directed abiogenesis is a project that is all that significant in terms of human knowledge, or human capability. What we
    would learn is how much of the cell's contents are essential for
    kickstarting life.

    I've often wondered how diverse the earth biosphere could have become if the final stages of the "protein takeover" had not taken place, but stopped with ribozymes being the main catalysts rather than protein enzymes.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of South Carolina in Columbia
    https://people.math.sc.edu/nyikos


    --
    alias Ernest Major

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Lawyer Daggett@21:1/5 to [email protected] on Wed Nov 1 02:52:04 2023
    On Wednesday, October 11, 2023 at 8:51:05 PM UTC-4, [email protected] wrote:
    On Tuesday, October 10, 2023 at 1:46:04 PM UTC-4, Lawyer Daggett still can't
    get the hang of what makes for a good chez watt:

    In the category of a case of begging the question being mistaken for a Chez Watt:
    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where
    new species came from.
    "IDers" is false. Even if the more nearly true word " creationists" is substituted, there are plenty of them who allow for new species or even new genera
    to evolve, by calling it "microevolution." The family Equidae, with tight sequences of
    genera, forces the more intelligent ones into this group.

    The IDer Behe, who has argued *for evolution* (more than I've seen Daggett do)
    goes a step further than most in _Darwin Devolves_. He considers a
    great deal of evolution as being due to breaking genes.

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    If Daggett had done some "spying" to see where the strengths and weaknesses of IDers lay, he would have seen an article in Evolution News
    about these broken genes.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of South Carolina
    http://people.math.sc.edu/nyikos

    Now that the month is over, I'm curious if you actually know who wrote
    "Note: we do not find IDers searching for answers as to how or where
    new species came from." and what the context was, and for that matter,
    how it has evolved. Or are you distracted by the pyramids?

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to Lawyer Daggett on Wed Nov 1 11:47:35 2023
    On Wednesday, November 1, 2023 at 5:56:27 AM UTC-4, Lawyer Daggett wrote:
    On Wednesday, October 11, 2023 at 8:51:05 PM UTC-4, [email protected] wrote:
    On Tuesday, October 10, 2023 at 1:46:04 PM UTC-4, Lawyer Daggett still can't
    get the hang of what makes for a good chez watt:

    In the category of a case of begging the question being mistaken for a Chez Watt:
    In the category of, yes, yes, and ...

    Note: we do not find IDers searching for answers as to how or where new species came from.

    "IDers" is false. Even if the more nearly true word " creationists" is substituted, there are plenty of them who allow for new species or even new genera
    to evolve, by calling it "microevolution." The family Equidae, with tight sequences of
    genera, forces the more intelligent ones into this group.

    The IDer Behe, who has argued *for evolution* (more than I've seen Daggett do)
    goes a step further than most in _Darwin Devolves_. He considers a
    great deal of evolution as being due to breaking genes.

    For instance, the woolly mammoth
    has been confirmed as having had close to a hundred genes broken
    that held it back from being adapted to very cold weather.

    If Daggett had done some "spying" to see where the strengths and weaknesses
    of IDers lay, he would have seen an article in Evolution News
    about these broken genes.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    University of South Carolina
    http://people.math.sc.edu/nyikos

    Now that the month is over, I'm curious if you actually know who wrote "Note: we do not find IDers searching for answers as to how or where
    new species came from." and what the context was, and for that matter,
    how it has evolved.

    I don't know for sure, but part of what makes Chez Watt relatively stress-free is that the anonymity the nominee is upheld as much as is feasible.
    I made some very critical remarks in the text above, I wish to spare the nominee any
    (further?) embarrassment.

    Or are you distracted by the pyramids?

    What do you mean by "pyramids"? The Israeli retaliation
    against Hamas is far removed from the pyramids.
    So you must be referring to something else.


    Peter Nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to All on Wed Nov 1 13:49:03 2023
    On Saturday, October 28, 2023 at 3:31:22 AM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Friday, October 27, 2023 at 3:41:21 PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20 PM UTC-4, jillery wrote: >>>> On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside
    from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such a way that the triplet to which it belongs still codes for the same amino acid.
    Is that what you were referring to as the predominate mode?

    Or a duplicate gene degrades while the original still remains functional.
    Or retroviruses and transposons litter the genome and remain as fossils. There are multiple ways junk accumulates.

    But not all junk is neutral. Some may be detrimental -- most birds have little of it,
    because of the excess weight involved (although ratites and other flightless birds may be an exception. Some may be beneficial, since "junk" may just
    mean "does not code for a polypeptide", but that is not the only function RNA has.
    [It just now occurred to me, though: might "junk DNA" include "does not undergo transcription"?]

    The point mutational view of
    evolution is a bit blinkered.

    I'll take that as a "no" answer to my question of what you meant by "predominate mode."


    Devolution conjures the dichotomy of the Morlocks and Eloi reflecting a
    turn of the century obsession with degeneration.

    https://www.merriam-webster.com/dictionary/devolve

    “…to degenerate through a gradual change or evolution”

    Reminds me of popular fears of inbreds that inspire horror movies like “Wrong Turn”.

    Would your prefer "burning their bridges behind them"? That seems to
    be Behe's main point.

    Degradation of genes from an adaptive state such as with our remnant
    yolking genes is not devolution but merely evolution.

    That's at the opposite extreme from the kinds of devolution that
    concerned Behe. Our "yolk sac" still gives many benefits even
    though we have a misnomer on our hands. The alternative term
    "umbilical vesicle" has been around for about a century now,
    and the late great human embryologist Ronan O'Rahilly
    has said it is about time to retire the term "yolk sac" for us humans.

    You can read a lot about the fine points of what this structure does
    for us humans and other mammals here:

    "Placentation in mammals: Definitive placenta, yolk sac, and paraplacenta" https://www.sciencedirect.com/science/article/pii/S0093691X16300802

    Here is an interesting excerpt which suggests that the "bridge burning"
    may have begun 200 million years or more ago:
    "Because they lay eggs, one might suppose monotremes to lack placentation. Yet two-thirds of embryonic development takes place in the uterus [76], and the embryo is nourished in part by endometrial secretions. These are taken up by the yolk sac through
    the egg shell membrane, which is porous and able to stretch as the embryo grows in size. This state of affairs is best described as matrotrophy [77], although it has been argued that the yolk sac of monotremes ought to be regarded as a placenta [78]."


    Are we devolved from
    our egg-laying ancestors because we don’t yolk our eggs?

    Some transhumanists of the future might think so, and might
    even endeavor to establish an extraterrestrial colony of oviparous descendants of humans, taking very seriously the slogans
    "pregnancy is a disease" and "abortion is healthcare."

    But I think that therian mammals (marsiupials and us eutherians)
    have more than compensated for that loss by our viviparity,
    which is developed in Homo sapiens further than in most eutherians.
    At this stage, an artificial uterus would probably result in the similar psychological dysfunctions to those seen in rhesus monkeys who had been "reared" by artificial "mothers" in a much-publicized experiment.


    That is incompatible with the breaking of a gene, which not only prevents it
    from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the events that transformed an elephant into a mammoth, that adapted the animal to its changing environment. A job well done, yes, but now those genes are gone forever, unavailable to help with the next change of environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Degradation of yolking genes in our ancestors should have done them in.

    Don't be so melodramatic. Unlike climate change, there doesn't seem to be
    any environmental change that would be seriously deleterious
    to viviparous mammals as opposed to our oviparous counterparts, the birds.

    Yet here we are with those broken genes. Amazing!

    Ridiculous. The issue is, what did we lose by having this or that breakage?
    It varies tremendously.

    I'd be more concerned by the loss of many regenerative capacities.
    Some urodeles can regrow whole limbs, but reptiles can only seem
    to regenerate tails, and the bones don't grow back in the new tail.
    Related are "generative" capacities that allow for increasing the
    number of vertebrae. The giant sauropods added lots of vertebrae
    to their necks, but even the giraffe is stuck with just 7, like almost
    all mammals. The edentates with a few extra can't even make the extras
    fit in as well as the sauropods could.

    Jillery took the words "gone forever" literally, but with the extinction of the woolly mammoth,
    it's a moot point. There are some who hope to resurrect mammoths by cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because
    the germ cell into which the nucleus of a mammoth cell is placed has an effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical mammoth on our hands.

    Ah yes epigenetics and the EES.

    I haven't heard much about the EES since the first flurry of excitement
    a half a decade or more ago. Have you caught wind of any new developments?


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of So. Carolina at Columbia
    http://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From Ernest Major@21:1/5 to [email protected] on Wed Nov 1 22:18:10 2023
    On 01/11/2023 20:49, [email protected] wrote:
    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such >>> a way that the triplet to which it belongs still codes for the same amino acid.

    I subscribe to nearly neutral evolution rather than neutral evolution.
    (For selection coefficients below a population size dependent value
    drift overwhelms selection.) Even synonymous changes to base pairs may
    have an associated selection coefficient.

    * it might change the energetic cost of replication
    * it might change the energetic cost of transcription
    * it might change the energetic cost of protein synthesis
    * the mutant may be more or less liable to give rise to a non-synonymous mutation - for example if it creates a CpG pair.
    * it might have an effective on DNA regulation, by changing the number
    of methylation sites in a gene, or by creating a spurious promoter site.
    * it might have an effect on protein synthesis by changing the rate the corresponding residue is added.

    In principle the various factors may cancel to give a selection
    coefficient of zero (rather than negligible), but since selection
    coefficients are real numbers I think that can be discounted.

    For the first three my intuition is that even in bacterial populations
    the selection coefficient is too small to be effectively seen by natural selection. But vertebrate genomes have a significant underrepresentation
    of CpG dinucleotides, which would seem to imply selection is at least
    strong enough to introduce a bias at least averaged across the genome.
    In the case of the last, I recall reading of instances where a change
    which results in using a commoner, or rarer, tRNA has the effect of
    changing the rate at which that step of translation occurs and changes
    the fidelity of protein folding.

    A base substitution in junk DNA is closer to a neutral change, but a
    some of the above factors still apply.

    Is that what you were referring to as the predominate mode?

    Usually when people refer to neutral evolution as the predominate mode
    they are referring to neutral evolution as the predominant source of
    genetic changes as opposed to adaptive selection, rather than to a
    particular class of mutations. (One might add stabilising and disruptive selection, or might argue that stabilising selection is source of
    non-evolution rather than evolution, and disruptive selection is a form
    of adaptive selection.)

    Or a duplicate gene degrades while the original still remains functional.
    Or retroviruses and transposons litter the genome and remain as fossils.
    There are multiple ways junk accumulates.
    But not all junk is neutral. Some may be detrimental -- most birds have little of it,
    because of the excess weight involved (although ratites and other flightless birds may be an exception.

    If junk is sufficiently detrimental it would get selected out. Junk is
    nearly neutral. I would expect that on average junk is very slightly detrimental due to metabolic cost of replication. Though there might be complications such as removing too much junk breaks the coordination of
    the cell cycle. (On the other hand the viability of polyploid cells
    implies that is not an issue at least most of the time.) Common wisdom
    is that with their large populations and short generation times the
    metabolic cost is large enough to be under selection in bacteria, with
    the result that they have little junk DNA, but in eukaryotes with longer
    cell cycle times and smaller populations the metabolic cost flies under
    the radar.

    The story I recall about birds (and which comes up on a web search) is
    that it's not the weight of the DNA that's the issue, but that birds
    have smaller cells. The web tells me that the smaller cells support the metabolic costs of flight by improving oxygen intake (because of the square-cube law); how this translates to less DNA was not made explicit
    but it might be that a smaller nucleus means more room for mitochondria.

    Some may be beneficial, since "junk" may just
    mean "does not code for a polypeptide", but that is not the only function RNA has.

    No, junk does not mean "does not code for a polypeptide" - that's a panadaptionist strawman.

    [It just now occurred to me, though: might "junk DNA" include "does not undergo transcription"?]

    No, junk does not mean "does not undergo transcription", on both sides
    of the coin. On the one hand centromeres, telomeres and regulatory
    binding sites inter alia do not under the normal course of events
    undergo transcription. On the other hand, introns do undergo
    transcription, and there is a low level of transcriptional noise across
    most of the genome.

    You might benefit from reading Larry Moran's new book on the genome.

    --
    alias Ernest Major

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From John Harshman@21:1/5 to Ernest Major on Wed Nov 1 16:59:59 2023
    On 11/1/23 3:18 PM, Ernest Major wrote:
    On 01/11/2023 20:49, [email protected] wrote:
    If by "neutral evolution" you mean no effect on the fitness of the
    organism,
    the clearest form is when a nucleotide is replaced by another one in
    such
    a way that the triplet to which it belongs still codes for the same
    amino acid.

    I subscribe to nearly neutral evolution rather than neutral evolution.
    (For selection coefficients below a population size dependent value
    drift overwhelms selection.) Even synonymous changes to base pairs may
    have an associated selection coefficient.

    * it might change the energetic cost of replication
    * it might change the energetic cost of transcription
    * it might change the energetic cost of protein synthesis
    * the mutant may be more or less liable to give rise to a non-synonymous mutation - for example if it creates a CpG pair.
    * it might have an effective on DNA regulation, by changing the number
    of methylation sites in a gene, or by creating a spurious promoter site.
    * it might have an effect on protein synthesis by changing the rate the corresponding residue is added.

    In principle the various factors may cancel to give a selection
    coefficient of zero (rather than negligible), but since selection coefficients are real numbers I think that can be discounted.

    For the first three my intuition is that even in bacterial populations
    the selection coefficient is too small to be effectively seen by natural selection. But vertebrate genomes have a significant underrepresentation
    of CpG dinucleotides, which would seem to imply selection is at least
    strong enough to introduce a bias at least averaged across the genome.
    In the case of the last, I recall reading of instances where a change
    which results in using a commoner, or rarer, tRNA has the effect of
    changing the rate at which that step of translation occurs and changes
    the fidelity of protein folding.

    A base substitution in junk DNA is closer to a neutral change, but a
    some of the above factors still apply.

    Is that what you were referring to as the predominate mode?

    Usually when people refer to neutral evolution as the predominate mode
    they are referring to neutral evolution as the predominant source of
    genetic changes as opposed to adaptive selection, rather than to a
    particular class of mutations. (One might add stabilising and disruptive selection, or might argue that stabilising selection is source of non-evolution rather than evolution, and disruptive selection is a form
    of adaptive selection.)

    Or a duplicate gene degrades while the original still remains
    functional.
    Or retroviruses and transposons litter the genome and remain as fossils. >>> There are multiple ways junk accumulates.
    But not all junk is neutral. Some may be detrimental -- most birds
    have little of it,
    because of the excess weight involved (although ratites and other
    flightless
    birds may be an exception.

    If junk is sufficiently detrimental it would get selected out. Junk is
    nearly neutral. I would expect that on average junk is very slightly detrimental due to metabolic cost of replication. Though there might be complications such as removing too much junk breaks the coordination of
    the cell cycle. (On the other hand the viability of polyploid cells
    implies that is not an issue at least most of the time.) Common wisdom
    is that with their large populations and short generation times the
    metabolic cost is large enough to be under selection in bacteria, with
    the result that they have little junk DNA, but in eukaryotes with longer
    cell cycle times and smaller populations the metabolic cost flies under
    the radar.

    The story I recall about birds (and which comes up on a web search) is
    that it's not the weight of the DNA that's the issue, but that birds
    have smaller cells. The web tells me that the smaller cells support the metabolic costs of flight by improving oxygen intake (because of the square-cube law); how this translates to less DNA was not made explicit
    but it might be that a smaller nucleus means more room for mitochondria.

    Two points: First, birds have plenty of junk. The average bird genome is
    about 2/3 the size of the average mammal genome, but that's still mostly
    junk. Second, apparently the major reduction in dinosaur genome size
    (and thus cell size) happened before flight. I recall a paper in Nature
    to that effect.

    https://www.nature.com/articles/nature05621

    Some may be beneficial, since "junk" may just
    mean "does not code for a polypeptide", but that is not the only
    function RNA has.

    No, junk does not mean "does not code for a polypeptide" - that's a panadaptionist strawman.

      [It just now occurred to me, though: might "junk DNA" include "does
    not undergo transcription"?]

    No, junk does not mean "does not undergo transcription", on both sides
    of the coin. On the one hand centromeres, telomeres and regulatory
    binding sites inter alia do not under the normal course of events
    undergo transcription. On the other hand, introns do undergo
    transcription, and there is a low level of transcriptional noise across
    most of the genome.

    You might benefit from reading Larry Moran's new book on the genome.

    Title: "What's in Your Genome?"

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to Ernest Major on Wed Nov 1 16:45:41 2023
    On Wednesday, November 1, 2023 at 6:21:26 PM UTC-4, Ernest Major wrote:
    On 01/11/2023 20:49, [email protected] wrote:
    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such
    a way that the triplet to which it belongs still codes for the same amino acid.
    I subscribe to nearly neutral evolution rather than neutral evolution.
    (For selection coefficients below a population size dependent value
    drift overwhelms selection.) Even synonymous changes to base pairs may
    have an associated selection coefficient.

    * it might change the energetic cost of replication
    * it might change the energetic cost of transcription
    * it might change the energetic cost of protein synthesis
    * the mutant may be more or less liable to give rise to a non-synonymous mutation - for example if it creates a CpG pair.
    * it might have an effective on DNA regulation, by changing the number
    of methylation sites in a gene, or by creating a spurious promoter site.
    * it might have an effect on protein synthesis by changing the rate the corresponding residue is added.

    The transfer RNAs corresponding to synonymous triplets are not present in equal concentration, so a synonymous mutation can change the rate at which translation occurs (because you might have to wait for a relatively rare tRNA). Pauses in the right spot
    during translation can lead to misfolding as well. When you try to express a gene from one species in cells from another, the level of expression is often increased if you rewrite the original gene using the synonymous codon biases of the second species.
    Also, synonymous changes can affect mRNA secondary structure, which in turn can affect mRNA half-life and thus the ultimate level of protein expression from the gene. So it is certainly plausible that synonymous changes may have a selectable effect.

    In principle the various factors may cancel to give a selection
    coefficient of zero (rather than negligible), but since selection coefficients are real numbers I think that can be discounted.

    For the first three my intuition is that even in bacterial populations
    the selection coefficient is too small to be effectively seen by natural selection. But vertebrate genomes have a significant underrepresentation
    of CpG dinucleotides, which would seem to imply selection is at least
    strong enough to introduce a bias at least averaged across the genome.
    In the case of the last, I recall reading of instances where a change
    which results in using a commoner, or rarer, tRNA has the effect of
    changing the rate at which that step of translation occurs and changes
    the fidelity of protein folding.

    A base substitution in junk DNA is closer to a neutral change, but a
    some of the above factors still apply.
    Is that what you were referring to as the predominate mode?
    Usually when people refer to neutral evolution as the predominate mode
    they are referring to neutral evolution as the predominant source of
    genetic changes as opposed to adaptive selection, rather than to a particular class of mutations. (One might add stabilising and disruptive selection, or might argue that stabilising selection is source of non-evolution rather than evolution, and disruptive selection is a form
    of adaptive selection.)

    Or a duplicate gene degrades while the original still remains functional. >> Or retroviruses and transposons litter the genome and remain as fossils. >> There are multiple ways junk accumulates.
    But not all junk is neutral. Some may be detrimental -- most birds have little of it,
    because of the excess weight involved (although ratites and other flightless
    birds may be an exception.
    If junk is sufficiently detrimental it would get selected out. Junk is nearly neutral. I would expect that on average junk is very slightly detrimental due to metabolic cost of replication. Though there might be complications such as removing too much junk breaks the coordination of
    the cell cycle. (On the other hand the viability of polyploid cells
    implies that is not an issue at least most of the time.) Common wisdom
    is that with their large populations and short generation times the metabolic cost is large enough to be under selection in bacteria, with
    the result that they have little junk DNA, but in eukaryotes with longer cell cycle times and smaller populations the metabolic cost flies under
    the radar.

    The story I recall about birds (and which comes up on a web search) is
    that it's not the weight of the DNA that's the issue, but that birds
    have smaller cells. The web tells me that the smaller cells support the metabolic costs of flight by improving oxygen intake (because of the square-cube law); how this translates to less DNA was not made explicit
    but it might be that a smaller nucleus means more room for mitochondria.
    Some may be beneficial, since "junk" may just
    mean "does not code for a polypeptide", but that is not the only function RNA has.
    No, junk does not mean "does not code for a polypeptide" - that's a panadaptionist strawman.
    [It just now occurred to me, though: might "junk DNA" include "does not undergo transcription"?]
    No, junk does not mean "does not undergo transcription", on both sides
    of the coin. On the one hand centromeres, telomeres and regulatory
    binding sites inter alia do not under the normal course of events
    undergo transcription. On the other hand, introns do undergo
    transcription, and there is a low level of transcriptional noise across
    most of the genome.

    You might benefit from reading Larry Moran's new book on the genome.

    --
    alias Ernest Major

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to {$to$}@meden.demon.co.uk on Thu Nov 2 11:14:56 2023
    On Tue, 31 Oct 2023 20:20:16 +0000, Ernest Major
    <{$to$}@meden.demon.co.uk> wrote:

    On 31/10/2023 19:03, [email protected] wrote:
    On Sunday, October 29, 2023 at 7:56:23?AM UTC-4, Ernest Major wrote:
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:

    the germ cell into which the nucleus of a mammoth cell is placed has an >>>>> effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical >>>>> mammoth on our hands.

    Ah yes epigenetics and the EES.

    The interaction between the mitome and nucleome isn't epigenetics.

    Thanks for the correction.


    The term that applies to traits that result from the interaction of
    different genes is polygenic.

    Is there a term which applies specifically to the interaction
    between the genes of the nucleus and those of the mitochondria?

    Not that I know of. (Cytoplasmic male sterility applies - assuming it's
    the mitochondria rather than the plastids that are involved - but that's >only one of many possible interactions.)


    A genuine mammoth would require being raised by genuine mammoth
    parents. The case described above would be at best a biological model
    of a mammoth.

    <snip-a-doodle>

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Thu Nov 2 12:01:51 2023
    On Tue, 31 Oct 2023 12:34:30 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Saturday, October 28, 2023 at 12:46:22?AM UTC-4, jillery wrote:
    On Fri, 27 Oct 2023 14:25:42 -0700 (PDT), "[email protected]"
    <[email protected]> wrote:
    On Friday, October 27, 2023 at 3:41:21?PM UTC-4, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:
    On Thursday, October 26, 2023 at 12:16:20?PM UTC-4, jillery wrote:
    On Thu, 26 Oct 2023 10:05:34 +0000, *Hemidactylus*
    <[email protected]d> wrote:

    Crickets chirping in the gentle topical breeze.

    'Tis Hawaiian crickets, proof of devolution but contrary to ID.

    Not contrary, just compatible with the absence of ID.

    Is the "devolution" part correct? That is, did Hawaiian crickets
    devolve from their ancestors due to the breaking of genes?

    By "devolve" is meant that the evolutionary changes were due to the
    degradation of the genome.

    How is that different from the predominate mode of neutral evolution aside
    from being an implicit value judgment?

    If by "neutral evolution" you mean no effect on the fitness of the organism,
    the clearest form is when a nucleotide is replaced by another one in such >> >a way that the triplet to which it belongs still codes for the same amino acid.
    Is that what you were referring to as the predominate mode?

    That is incompatible with the breaking of a gene, which not only prevents it
    from coding for the same polypeptide, it even prevents it from coding for anything.
    Behe put it colorfully and informally about mammoth genes:

    "The point is that these gene losses aren’t side shows — they are the events that transformed an elephant into a mammoth, that adapted the animal to its changing environment. A job well done, yes, but now those genes are gone forever, unavailable
    to help with the next change of environment. Perhaps that contributed to eventual mammoth extinction."
    -- https://evolutionnews.org/2022/08/mammoth-support-for-devolution/

    Jillery took the words "gone forever" literally,

    To be accurate, Behe dishonestly exaggerated the facts.

    Not so fast. There is more than one gene involved here.


    Not the point. Whether one or many, they are unambiguously not "gone
    forever". Inactive genes remain in genomes until random chance
    removes them.


    Excerpt from linked article:
    In Darwin Devolves, I also mentioned work on DNA extracted from frozen woolly mammoth carcasses that showcased devolution: “26 genes were shown to be seriously degraded, many of which (as with polar bear) were involved in fat metabolism, critical in
    the extremely cold environments that the mammoth roamed.”
    [end of excerpt]

    Even if one of those genes somehow got resurrected from its broken
    state to its original state -- unlikely in the time from the first woollymammoth
    to historical times [1]-- the probability of it happening to the majority >of the relevant genes is so small as to make it a practical impossibility.

    [1] "once in a million blue moons" was the way I put it, and I don't think >that's an exaggeration


    You're entitled to your opinion, no matter how baseless. The issue
    here is not whether genes are "resurrected" to their original state,
    which is possible but unlikely. It's more likely they would
    "reincarnate" with different functions. Or they might move around and
    alter the expression of other genes.

    The point remains, Behe's argument is that genomes over time become
    more broken. This is suspiciously close to the Creationist PRATT that
    life was created perfect by God and has become corrupt over time due
    to Original Sin.


    Moreover, junk DNA alters more quickly than the genes that are
    expressed, due to the effects of natural selection not applying to them.
    If one of the broken ones did come to be expressed again,
    it could be that it would no longer code for its original polypeptide,
    but for another one that is not useful for the purpose of suppressing
    the amount of insulating fat. This would put the individual at
    a disadvantage in times of global warming.

    but with the extinction of the woolly mammoth, it's a moot point.

    If so, then mammoth extinction would also moot Behe's entire line of
    reasoning.

    It was quite strong while the mammoth was in existence.


    Then make up your mind which "it" and which point in time you're
    talking about.


    And it still applies to polar bears, another species discussed
    by Behe in the linked article. Also to musk oxen and other cold-adapted mammals.


    There are mutations which cause a loss of function. There are also
    mutations which alter the expression of functions to a greater or
    lesser degree. I noted one example elsethread about peppered moths.


    There are some who hope to resurrect mammoths by cloning, which would "unmoot" it,
    but the epigenetics of the situation make us unsure of success. That's because
    the germ cell into which the nucleus of a mammoth cell is placed has an effect on which genes
    are expressed. The embryonic development is influenced by the mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical mammoth on our hands.

    What you allude to above is a point I made previously about Dolly the
    sheep "clone" which was more accurately a chimera.

    Another problem with resurrecting extinct species is there are no
    parents to teach these organisms how to act appropriately. For these
    reasons and others, these experiments produce approximate models of
    extinct species.

    Good points.

    --
    To know less than we don't know is the nature of most knowledge

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From [email protected]@21:1/5 to jillery on Fri Nov 3 14:06:41 2023
    On Thursday, November 2, 2023 at 11:16:27 AM UTC-4, jillery wrote:
    On Tue, 31 Oct 2023 20:20:16 +0000, Ernest Major
    <{$to$}@meden.demon.co.uk> wrote:

    On 31/10/2023 19:03, [email protected] wrote:
    On Sunday, October 29, 2023 at 7:56:23?AM UTC-4, Ernest Major wrote:
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:

    the germ cell into which the nucleus of a mammoth cell is placed has an
    effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical >>>>> mammoth on our hands.

    Ah yes epigenetics and the EES.

    The interaction between the mitome and nucleome isn't epigenetics.

    Thanks for the correction.


    The term that applies to traits that result from the interaction of
    different genes is polygenic.

    Is there a term which applies specifically to the interaction
    between the genes of the nucleus and those of the mitochondria?

    Not that I know of. (Cytoplasmic male sterility applies - assuming it's >the mitochondria rather than the plastids that are involved - but that's >only one of many possible interactions.)

    A genuine mammoth would require being raised by genuine mammoth
    parents.

    Why? might not the habits of Asian elephants [the nearest extant relatives of the
    woolly mammoths] be close enough to the habits of mammoths to make
    the social interaction of the "resurrected" mammoths with the other members of the herd
    to be within normal bounds for each species?

    I'm assuming that the hypothetical researchers who produce a mammoth zygote and implant it into an elephant uterus can pick a suitable surrogate mother from a normal wild herd.


    The case described above would be at best a biological model
    of a mammoth.

    Depending on your (sketchily described) standards on the one hand,
    and their family history of captivity on the other, zoo elephants
    and circus elephants may already be far enough from their wild ancestors, socially speaking, to be considered "at best a biological model of Asian elephants."

    Please explain more about your standards if you disagree with this assessment.


    Peter Nyikos
    Professor, Dept. of Mathematics -- standard disclaimer--
    U. of So. Carolina in Columbia
    https://people.math.sc.edu/nyikos

    --- SoupGate-Win32 v1.05
    * Origin: fsxNet Usenet Gateway (21:1/5)
  • From jillery@21:1/5 to [email protected] on Sat Nov 4 05:50:55 2023
    On Fri, 3 Nov 2023 14:06:41 -0700 (PDT), "[email protected]" <[email protected]> wrote:

    On Thursday, November 2, 2023 at 11:16:27?AM UTC-4, jillery wrote:
    On Tue, 31 Oct 2023 20:20:16 +0000, Ernest Major
    <{$to$}@meden.demon.co.uk> wrote:

    On 31/10/2023 19:03, [email protected] wrote:
    On Sunday, October 29, 2023 at 7:56:23?AM UTC-4, Ernest Major wrote:
    On 28/10/2023 08:26, *Hemidactylus* wrote:
    [email protected] <[email protected]> wrote:

    the germ cell into which the nucleus of a mammoth cell is placed has an
    effect on which genes
    are expressed. The embryonic development is influenced by the
    mitochondria of the "mother" elephant,
    and that means that it's problematic that we would have a phenotypical
    mammoth on our hands.

    Ah yes epigenetics and the EES.

    The interaction between the mitome and nucleome isn't epigenetics.

    Thanks for the correction.


    The term that applies to traits that result from the interaction of
    different genes is polygenic.

    Is there a term which applies specifically to the interaction
    between the genes of the nucleus and those of the mitochondria?

    Not that I know of. (Cytoplasmic male sterility applies - assuming it's
    the mitochondria rather than the plastids that are involved - but that's >> >only one of many possible interactions.)

    A genuine mammoth would require being raised by genuine mammoth
    parents.

    Why? might not the habits of Asian elephants [the nearest extant relatives of the
    woolly mammoths] be close enough to the habits of mammoths to make
    the social interaction of the "resurrected" mammoths with the other members of the herd
    to be within normal bounds for each species?


    By your own words, you recognize that your case would raise clones
    with the habits of Asian elephants, and not with the habits of woolly
    mammoths. They would be approximate models and so not genuine.


    I'm assuming that the hypothetical researchers who produce a mammoth zygote >and implant it into an elephant uterus can pick a suitable surrogate mother >from a normal wild herd.


    The case described above would be at best a biological model
    of a mammoth.

    Depending on your (sketchily described) standards on the one hand,
    and their family history of captivity on the other, zoo elephants
    and circus elephants may already be far enough from their wild ancestors, >socially speaking, to be considered "at best a biological model of Asian elephants."

    Please explain more about your standards if you disagree with this assessment.


    My standards, sketchy or otherwise, don't inform any relevant
    discussion.

    --
    To know less than we don't know is the nature of most knowledge

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